| pubmed-article:11786644 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C1332717 | lld:lifeskim |
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| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C1413244 | lld:lifeskim |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C2698600 | lld:lifeskim |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C1420610 | lld:lifeskim |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C1423842 | lld:lifeskim |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
| pubmed-article:11786644 | lifeskim:mentions | umls-concept:C1881379 | lld:lifeskim |
| pubmed-article:11786644 | pubmed:issue | 5553 | lld:pubmed |
| pubmed-article:11786644 | pubmed:dateCreated | 2002-1-11 | lld:pubmed |
| pubmed-article:11786644 | pubmed:abstractText | T-bet is a member of the T-box family of transcription factors that appears to regulate lineage commitment in CD4 T helper (TH) lymphocytes in part by activating the hallmark TH1 cytokine, interferon-gamma (IFN-gamma). IFN-gamma is also produced by natural killer (NK) cells and most prominently by CD8 cytotoxic T cells, and is vital for the control of microbial pathogens. Although T-bet is expressed in all these cell types, it is required for control of IFN-gamma production in CD4 and NK cells, but not in CD8 cells. This difference is also apparent in the function of these cell subsets. Thus, the regulation of a single cytokine, IFN-gamma, is controlled by distinct transcriptional mechanisms within the T cell lineage. | lld:pubmed |
| pubmed-article:11786644 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11786644 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:11786644 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11786644 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11786644 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:11786644 | pubmed:issn | 1095-9203 | lld:pubmed |
| pubmed-article:11786644 | pubmed:author | pubmed-author:GlimcherLauri... | lld:pubmed |
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| pubmed-article:11786644 | pubmed:author | pubmed-author:SleckmanBarry... | lld:pubmed |
| pubmed-article:11786644 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:11786644 | pubmed:day | 11 | lld:pubmed |
| pubmed-article:11786644 | pubmed:volume | 295 | lld:pubmed |
| pubmed-article:11786644 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11786644 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11786644 | pubmed:pagination | 338-42 | lld:pubmed |
| pubmed-article:11786644 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:11786644 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:11786644 | pubmed:articleTitle | Distinct effects of T-bet in TH1 lineage commitment and IFN-gamma production in CD4 and CD8 T cells. | lld:pubmed |
| pubmed-article:11786644 | pubmed:affiliation | Department of Immunology and Infectious Diseases, Harvard School of Public Health, Boston, MA 02115, USA. | lld:pubmed |
| pubmed-article:11786644 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11786644 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11786644 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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