11709009

Source:http://linkedlifedata.com/resource/pubmed/id/11709009

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0010031, umls-concept:C0016781, umls-concept:C0035820, umls-concept:C0162638, umls-concept:C0699748
pubmed:issue	11
pubmed:dateCreated	2001-11-15
pubmed:abstractText	To investigate the potential role of apoptosis in the pathogenesis of Fuchs endothelial dystrophy of the cornea.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/7706534
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD95, http://linkedlifedata.com/resource/pubmed/chemical/BAX protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Camptothecin, http://linkedlifedata.com/resource/pubmed/chemical/DNA, http://linkedlifedata.com/resource/pubmed/chemical/DNA Primers, http://linkedlifedata.com/resource/pubmed/chemical/Enzyme Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/FASLG protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Fas Ligand Protein, http://linkedlifedata.com/resource/pubmed/chemical/Membrane Glycoproteins, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins c-bcl-2, http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger, http://linkedlifedata.com/resource/pubmed/chemical/Topoisomerase I Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/bcl-2-Associated X Protein
pubmed:status	MEDLINE
pubmed:month	Nov
pubmed:issn	0003-9950
pubmed:author	pubmed-author:AshrafM FMF, pubmed-author:ChanC CCC, pubmed-author:GreenW RWR, pubmed-author:LiQ JQJ, pubmed-author:O'BrienT PTP, pubmed-author:SheaD ADA, pubmed-author:StarkW JWJ
pubmed:issnType	Print
pubmed:volume	119
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	1597-604
pubmed:dateRevised	2010-11-18
pubmed:meshHeading	pubmed-meshheading:11709009-Aged, pubmed-meshheading:11709009-Aged, 80 and over, pubmed-meshheading:11709009-Antigens, CD95, pubmed-meshheading:11709009-Apoptosis, pubmed-meshheading:11709009-Camptothecin, pubmed-meshheading:11709009-Cells, Cultured, pubmed-meshheading:11709009-Corneal Stroma, pubmed-meshheading:11709009-DNA, pubmed-meshheading:11709009-DNA Primers, pubmed-meshheading:11709009-Enzyme Inhibitors, pubmed-meshheading:11709009-Fas Ligand Protein, pubmed-meshheading:11709009-Fibroblasts, pubmed-meshheading:11709009-Fuchs' Endothelial Dystrophy, pubmed-meshheading:11709009-Humans, pubmed-meshheading:11709009-Immunoenzyme Techniques, pubmed-meshheading:11709009-In Situ Nick-End Labeling, pubmed-meshheading:11709009-Membrane Glycoproteins, pubmed-meshheading:11709009-Middle Aged, pubmed-meshheading:11709009-Proto-Oncogene Proteins, pubmed-meshheading:11709009-Proto-Oncogene Proteins c-bcl-2, pubmed-meshheading:11709009-RNA, Messenger, pubmed-meshheading:11709009-Topoisomerase I Inhibitors, pubmed-meshheading:11709009-bcl-2-Associated X Protein
pubmed:year	2001
pubmed:articleTitle	The role of apoptosis in the pathogenesis of Fuchs endothelial dystrophy of the cornea.
pubmed:affiliation	Wilmer Eye Institute, The Johns Hopkins University School of Medicine, 600 N Wolfe St, Woods Bldg, Room 255, Baltimore, MD, USA.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't