| pubmed-article:11699080 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0006434 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0038435 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C1335960 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0699819 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0805732 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C1366753 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C1710082 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0312431 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0040661 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C1705162 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C0205548 | lld:lifeskim |
| pubmed-article:11699080 | lifeskim:mentions | umls-concept:C1548425 | lld:lifeskim |
| pubmed-article:11699080 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:11699080 | pubmed:dateCreated | 2001-11-8 | lld:pubmed |
| pubmed-article:11699080 | pubmed:abstractText | A burn injury triggers traumatic reactions characteristics of a stress. Here we investigated the early responses of prolactin (PRL), corticosterone (CS), and signal transducer and activator of transcription 5 (STAT5) in male Sprague-Dawley rats after burn injury. PRL and CS levels were determined in blood serum. STAT5 and phospho-STAT5 levels were determined in jejunum total protein extracts. The results confirmed an expected increase of CS between 4 and 6 h after the burn injury. Unexpectedly, PRL secretion was suppressed during the same time frame. These hormone levels returned to normal 6 to 8 h after burn injury. STAT5 was increased in the jejunum after burn injury, and its phosphorylation was increased between 8 and 11 h after burn injury. These changes in STAT5 were not temporally correlated with either the hormone changes that we observed or with previously documented changes of the gut function after burns. | lld:pubmed |
| pubmed-article:11699080 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11699080 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11699080 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11699080 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:11699080 | pubmed:issn | 1073-2322 | lld:pubmed |
| pubmed-article:11699080 | pubmed:author | pubmed-author:KhuranaSS | lld:pubmed |
| pubmed-article:11699080 | pubmed:author | pubmed-author:NoelGG | lld:pubmed |
| pubmed-article:11699080 | pubmed:author | pubmed-author:HorsemanN DND | lld:pubmed |
| pubmed-article:11699080 | pubmed:author | pubmed-author:OgleC KCK | lld:pubmed |
| pubmed-article:11699080 | pubmed:author | pubmed-author:ThellinOO | lld:pubmed |
| pubmed-article:11699080 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11699080 | pubmed:volume | 16 | lld:pubmed |
| pubmed-article:11699080 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11699080 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11699080 | pubmed:pagination | 393-7 | lld:pubmed |
| pubmed-article:11699080 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:meshHeading | pubmed-meshheading:11699080... | lld:pubmed |
| pubmed-article:11699080 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11699080 | pubmed:articleTitle | Stress hormone secretion and gut signal transducer (STAT) proteins after burn injury in rats. | lld:pubmed |
| pubmed-article:11699080 | pubmed:affiliation | Department of Molecular and Cellular Physiology, University of Cincinnati, 231 Bethesda Avenue, Cincinnati, OH 45267-0576, USA. | lld:pubmed |
| pubmed-article:11699080 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11699080 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11699080 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
