Linked Life Data

11677282

Source:http://linkedlifedata.com/resource/pubmed/id/11677282

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
913
pubmed:dateCreated
2001-10-25
pubmed:abstractText
The clinical syndrome of encephalopathy is most often encountered in the context of decompensated liver disease and the diagnosis is usually clear cut. Non-hepatic causes of encephalopathy are rarer and tend to present to a wide range of medical specialties with variable and episodic symptoms. Delay can result in the development of potentially life threatening complications, such as seizures and coma. Early recognition is vital. A history of similar episodes or clinical risk factors and early assessment of blood ammonia levels help establish the diagnosis. In addition to adequate supportive care, investigation of the underlying cause of the hyperammonaemia is essential and its reversal, where possible, will often result in complete recovery. Detection of an unborn error of metabolism should lead to the initiation of appropriate maintenance therapy and genetic counselling.
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
0032-5473
pubmed:author
pubmed:issnType
Print
pubmed:volume
77
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
717-22
pubmed:dateRevised
2009-10-22
pubmed:meshHeading
pubmed:year
2001
pubmed:articleTitle
Non-hepatic hyperammonaemia: an important, potentially reversible cause of encephalopathy.
pubmed:affiliation
Department of Gastroenterology, University Hospital of Wales, Heath Park, Cardiff, UK. neihawkes@doctors.org.uk
pubmed:publicationType
Journal Article, Case Reports