| pubmed-article:11544291 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11544291 | lifeskim:mentions | umls-concept:C1257890 | lld:lifeskim |
| pubmed-article:11544291 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:11544291 | lifeskim:mentions | umls-concept:C0030664 | lld:lifeskim |
| pubmed-article:11544291 | lifeskim:mentions | umls-concept:C0004368 | lld:lifeskim |
| pubmed-article:11544291 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
| pubmed-article:11544291 | lifeskim:mentions | umls-concept:C0439828 | lld:lifeskim |
| pubmed-article:11544291 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:11544291 | pubmed:dateCreated | 2001-9-6 | lld:pubmed |
| pubmed-article:11544291 | pubmed:abstractText | It has become increasingly apparent in studies of mutant mice and observations of disease that cytokine production by fully committed effector T cells within the Th1 and Th2 phenotype can vary within each group. This can potentially influence the type and effectiveness of a given immune response. The factors responsible for inducing variable Th1 and Th2 subtype responses have not been well established. Using transgenic mice expressing the myelin basic protein-specific TCR, we demonstrate here that two distinct populations of Th2 cells that are characterized primarily by differential IL-4 and IL-5 expression levels can be generated depending upon the levels of IFN-gamma present at the time of priming. We also demonstrate that populations expressing high levels of IL-4 relative to IL-5 vs those with intermediate levels of IL-4 relative to IL-5 are stable and possess distinct effector functions in an experimental autoimmune encephalomyelitis model. | lld:pubmed |
| pubmed-article:11544291 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11544291 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11544291 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:11544291 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11544291 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:11544291 | pubmed:issn | 0022-1767 | lld:pubmed |
| pubmed-article:11544291 | pubmed:author | pubmed-author:LafailleJ JJJ | lld:pubmed |
| pubmed-article:11544291 | pubmed:author | pubmed-author:MarcondesM... | lld:pubmed |
| pubmed-article:11544291 | pubmed:author | pubmed-author:WenskyAA | lld:pubmed |
| pubmed-article:11544291 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11544291 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:11544291 | pubmed:volume | 167 | lld:pubmed |
| pubmed-article:11544291 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11544291 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11544291 | pubmed:pagination | 3074-81 | lld:pubmed |
| pubmed-article:11544291 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:11544291 | pubmed:meshHeading | pubmed-meshheading:11544291... | lld:pubmed |
| pubmed-article:11544291 | pubmed:meshHeading | pubmed-meshheading:11544291... | lld:pubmed |
| pubmed-article:11544291 | pubmed:meshHeading | pubmed-meshheading:11544291... | lld:pubmed |
| pubmed-article:11544291 | pubmed:meshHeading | pubmed-meshheading:11544291... | lld:pubmed |
| pubmed-article:11544291 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11544291 | pubmed:articleTitle | The role of IFN-gamma in the production of Th2 subpopulations: implications for variable Th2-mediated pathologies in autoimmunity. | lld:pubmed |
| pubmed-article:11544291 | pubmed:affiliation | Division of Molecular Pathogenesis, Skirball Institute of Biomolecular Medicine, Department of Pathology, New York University Medical Center, New York, NY 10016, USA. | lld:pubmed |
| pubmed-article:11544291 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11544291 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11544291 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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