| pubmed-article:11525733 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11525733 | lifeskim:mentions | umls-concept:C0162741 | lld:lifeskim |
| pubmed-article:11525733 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:11525733 | lifeskim:mentions | umls-concept:C0000843 | lld:lifeskim |
| pubmed-article:11525733 | lifeskim:mentions | umls-concept:C0073423 | lld:lifeskim |
| pubmed-article:11525733 | lifeskim:mentions | umls-concept:C1823174 | lld:lifeskim |
| pubmed-article:11525733 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:11525733 | pubmed:dateCreated | 2001-8-29 | lld:pubmed |
| pubmed-article:11525733 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11525733 | pubmed:abstractText | The plant hormone abscisic acid (ABA) regulates important stress and developmental responses. We have isolated a recessive ABA hypersensitive mutant, abh1, that shows hormone specificity to ABA. ABH1 encodes the Arabidopsis homolog of a nuclear mRNA cap binding protein and functions in a heterodimeric complex to bind the mRNA cap structure. DNA chip analyses show that only a few transcripts are down-regulated in abh1, several of which are implicated in ABA signaling. Consistent with these results, abh1 plants show ABA-hypersensitive stomatal closing and reduced wilting during drought. Interestingly, ABA-hypersensitive cytosolic calcium increases in abh1 guard cells demonstrate amplification of early ABA signaling. Thus, ABH1 represents a modulator of ABA signaling proposed to function by transcript alteration of early ABA signaling elements. | lld:pubmed |
| pubmed-article:11525733 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11525733 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:11525733 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11525733 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11525733 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:11525733 | pubmed:issn | 0092-8674 | lld:pubmed |
| pubmed-article:11525733 | pubmed:author | pubmed-author:SchroederJ... | lld:pubmed |
| pubmed-article:11525733 | pubmed:author | pubmed-author:HUMEA CAC | lld:pubmed |
| pubmed-article:11525733 | pubmed:author | pubmed-author:HugouvieuxVV | lld:pubmed |
| pubmed-article:11525733 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11525733 | pubmed:day | 24 | lld:pubmed |
| pubmed-article:11525733 | pubmed:volume | 106 | lld:pubmed |
| pubmed-article:11525733 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11525733 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11525733 | pubmed:pagination | 477-87 | lld:pubmed |
| pubmed-article:11525733 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:11525733 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11525733 | pubmed:articleTitle | An mRNA cap binding protein, ABH1, modulates early abscisic acid signal transduction in Arabidopsis. | lld:pubmed |
| pubmed-article:11525733 | pubmed:affiliation | Division of Biology, Cell and Developmental Biology Section, University of California San Diego, 9500 Gilman Drive, La Jolla, CA 92093, USA. | lld:pubmed |
| pubmed-article:11525733 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11525733 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11525733 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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