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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
8
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pubmed:dateCreated |
2001-8-16
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pubmed:abstractText |
Approximately 5% to 20% of fractures have delayed or impaired healing. Therefore, it is desirable to develop new therapies to enhance fracture-healing that can be used in conjunction with traditional treatment methods. The purpose of this study was to evaluate the ability of a single application of recombinant human bone morphogenetic protein-2 to accelerate fracture-healing in a rabbit ulnar osteotomy that heals spontaneously.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
0021-9355
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
83-A
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1219-30
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pubmed:dateRevised |
2010-10-25
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pubmed:meshHeading |
pubmed-meshheading:11507131-Animals,
pubmed-meshheading:11507131-Biomechanics,
pubmed-meshheading:11507131-Bone Density,
pubmed-meshheading:11507131-Bone Morphogenetic Protein 2,
pubmed-meshheading:11507131-Bone Morphogenetic Proteins,
pubmed-meshheading:11507131-Bony Callus,
pubmed-meshheading:11507131-Humans,
pubmed-meshheading:11507131-Male,
pubmed-meshheading:11507131-Models, Animal,
pubmed-meshheading:11507131-Osteotomy,
pubmed-meshheading:11507131-Rabbits,
pubmed-meshheading:11507131-Recombinant Proteins,
pubmed-meshheading:11507131-Transforming Growth Factor beta,
pubmed-meshheading:11507131-Ulna Fractures,
pubmed-meshheading:11507131-Wound Healing
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pubmed:year |
2001
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pubmed:articleTitle |
Recombinant human bone morphogenetic protein-2 accelerates healing in a rabbit ulnar osteotomy model.
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pubmed:affiliation |
Musculoskeletal Sciences, Genetics Institute/Wyeth-Ayerst Research, Cambridge, MA 02140, USA. mbouxsein@genetics.com
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pubmed:publicationType |
Journal Article
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