Linked Life Data

11452797

Source:http://linkedlifedata.com/resource/pubmed/id/11452797

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2001-7-16
pubmed:abstractText
I.v. administration of the delta-opioid (OR) receptors' agonists DSLET or DTLET prevented creatine kinase leakage from the rat isolated heart in oxidative stress damage and abolished an increase in myocardial levels of conjugated diens and malondialdehyde. The agonists also prevented a stress-induced augmentation of the superoxide dismutase (SOD) activity. All protective effects of delta-receptor stimulation was completely abolished by the delta OR antagonist ICI 174,864. The data obtained suggest that the cardioprotective effect of the delta OR stimulation in vivo is not mediated via direct cardiac delta OR activation but, probably, rather via some unknown indirect circulating humoral factor(s).
pubmed:language
rus
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
0869-8139
pubmed:author
pubmed:issnType
Print
pubmed:volume
87
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
628-41
pubmed:dateRevised
2008-8-22
pubmed:meshHeading
pubmed:year
2001
pubmed:articleTitle
[The role of mu- and delta-opiate receptors in resistance of the myocardium to free radical damage].
pubmed:affiliation
Institute of Cardiology, Kievskaya St., 111, 634050 Tomsk, Russia.
pubmed:publicationType
Journal Article, In Vitro, English Abstract