11435743

Source:http://linkedlifedata.com/resource/pubmed/id/11435743

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0028128, umls-concept:C0035820, umls-concept:C0086418, umls-concept:C0277785, umls-concept:C0599918
pubmed:issue	2
pubmed:dateCreated	2001-7-3
pubmed:abstractText	The causes for the nephrotoxicity of cyclosporine A (CsA) have not been fully elucidated. Intrarenal vasoconstriction induced by several different mediators, both in humans and experimental animals, have been proposed.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9610505
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/6-Ketoprostaglandin F1 alpha, http://linkedlifedata.com/resource/pubmed/chemical/Cyclosporine, http://linkedlifedata.com/resource/pubmed/chemical/Dinoprostone, http://linkedlifedata.com/resource/pubmed/chemical/Endothelins, http://linkedlifedata.com/resource/pubmed/chemical/Immunosuppressive Agents, http://linkedlifedata.com/resource/pubmed/chemical/Inulin, http://linkedlifedata.com/resource/pubmed/chemical/Nitrates, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide, http://linkedlifedata.com/resource/pubmed/chemical/Nitrites, http://linkedlifedata.com/resource/pubmed/chemical/Prostaglandins, http://linkedlifedata.com/resource/pubmed/chemical/Thromboxane B2, http://linkedlifedata.com/resource/pubmed/chemical/Vasoconstrictor Agents, http://linkedlifedata.com/resource/pubmed/chemical/p-Aminohippuric Acid
pubmed:status	MEDLINE
pubmed:issn	1420-4096
pubmed:author	pubmed-author:BernemannAA, pubmed-author:BickeböllerRR, pubmed-author:GossmannJJ, pubmed-author:RaabH PHP, pubmed-author:RadounikliAA, pubmed-author:SchellinskiOO, pubmed-author:ScheuermannE HEH
pubmed:issnType	Print
pubmed:volume	24
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	111-5
pubmed:dateRevised	2006-11-15
pubmed:meshHeading	pubmed-meshheading:11435743-6-Ketoprostaglandin F1 alpha, pubmed-meshheading:11435743-Administration, Oral, pubmed-meshheading:11435743-Adult, pubmed-meshheading:11435743-Blood Pressure, pubmed-meshheading:11435743-Cyclosporine, pubmed-meshheading:11435743-Dinoprostone, pubmed-meshheading:11435743-Endothelins, pubmed-meshheading:11435743-Female, pubmed-meshheading:11435743-Heart Rate, pubmed-meshheading:11435743-Humans, pubmed-meshheading:11435743-Immunosuppressive Agents, pubmed-meshheading:11435743-Inulin, pubmed-meshheading:11435743-Kidney Diseases, pubmed-meshheading:11435743-Kidney Transplantation, pubmed-meshheading:11435743-Male, pubmed-meshheading:11435743-Metabolic Clearance Rate, pubmed-meshheading:11435743-Nitrates, pubmed-meshheading:11435743-Nitric Oxide, pubmed-meshheading:11435743-Nitrites, pubmed-meshheading:11435743-Prostaglandins, pubmed-meshheading:11435743-Renal Artery, pubmed-meshheading:11435743-Renal Circulation, pubmed-meshheading:11435743-Thromboxane B2, pubmed-meshheading:11435743-Vascular Resistance, pubmed-meshheading:11435743-Vasoconstrictor Agents, pubmed-meshheading:11435743-p-Aminohippuric Acid
pubmed:year	2001
pubmed:articleTitle	Pathophysiology of cyclosporine-induced nephrotoxicity in humans: a role for nitric oxide?
pubmed:affiliation	Funktionsbereich Nephrologie, Medizinische Klinik IV, Klinikum der J.W.-Goethe-Universität, Frankfurt am Main, Deutschland.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't