Linked Life Data

11336187

Source:http://linkedlifedata.com/resource/pubmed/id/11336187

Statements in which the resource exists.
SubjectPredicateObjectContext
pubmed-article:11336187rdf:typepubmed:Citationlld:pubmed
pubmed-article:11336187lifeskim:mentionsumls-concept:C0020538lld:lifeskim
pubmed-article:11336187lifeskim:mentionsumls-concept:C0028128lld:lifeskim
pubmed-article:11336187lifeskim:mentionsumls-concept:C0011710lld:lifeskim
pubmed-article:11336187lifeskim:mentionsumls-concept:C0037494lld:lifeskim
pubmed-article:11336187lifeskim:mentionsumls-concept:C0813203lld:lifeskim
pubmed-article:11336187lifeskim:mentionsumls-concept:C0205216lld:lifeskim
pubmed-article:11336187lifeskim:mentionsumls-concept:C0000975lld:lifeskim
pubmed-article:11336187pubmed:issue4 Pt 1lld:pubmed
pubmed-article:11336187pubmed:dateCreated2001-5-4lld:pubmed
pubmed-article:11336187pubmed:abstractTextThe response to exogenous administration of acetylcholine and the effects of nitric oxide (NO) synthesis blockade were assessed for small arterioles in the cremaster muscle of deoxycorticosterone acetate (DOCA)-salt hypertensive and normotensive rats using intravital microscopy. The NO synthesis inhibitor N omega-L-nitro-arginine methyl ester (L-NAME) produced significantly less constriction in the arterioles of DOCA-salt hypertensive rats. Exposure to increasing concentrations of acetylcholine (10(-10) to 10(-5) mol/L) or sodium nitroprusside (10(-10) to 10(-5) mol/L) produced similar arteriolar dilation in both groups. These results suggest that attenuated basal release of NO by arterioles may play a role in the development of increased peripheral resistance observed in DOCA-salt hypertension.lld:pubmed
pubmed-article:11336187pubmed:languageenglld:pubmed
pubmed-article:11336187pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:citationSubsetIMlld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:11336187pubmed:statusMEDLINElld:pubmed
pubmed-article:11336187pubmed:monthAprlld:pubmed
pubmed-article:11336187pubmed:issn0895-7061lld:pubmed
pubmed-article:11336187pubmed:authorpubmed-author:JoshuaI GIGlld:pubmed
pubmed-article:11336187pubmed:authorpubmed-author:Ayangade-John...lld:pubmed
pubmed-article:11336187pubmed:issnTypePrintlld:pubmed
pubmed-article:11336187pubmed:volume14lld:pubmed
pubmed-article:11336187pubmed:ownerNLMlld:pubmed
pubmed-article:11336187pubmed:authorsCompleteYlld:pubmed
pubmed-article:11336187pubmed:pagination387-9lld:pubmed
pubmed-article:11336187pubmed:dateRevised2009-2-24lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:meshHeadingpubmed-meshheading:11336187...lld:pubmed
pubmed-article:11336187pubmed:year2001lld:pubmed
pubmed-article:11336187pubmed:articleTitleDecreased influence of nitric oxide on deoxycorticosterone acetate (DOCA)-salt hypertension.lld:pubmed
pubmed-article:11336187pubmed:affiliationDepartment of Pediatrics, University of Louisville School of Medicine, Kentucky, USA.lld:pubmed
pubmed-article:11336187pubmed:publicationTypeJournal Articlelld:pubmed