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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2001-3-27
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pubmed:abstractText |
Gene delivery to the myocardium using blood-borne adenoviral vectors is hindered by the endothelium, which represents a barrier limiting the infection rate of underlying myocytes. However, endothelial permeability may be modulated by pharmacological agents.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
1099-498X
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
3
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
42-50
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:11269335-Adenoviridae,
pubmed-meshheading:11269335-Animals,
pubmed-meshheading:11269335-Capillary Permeability,
pubmed-meshheading:11269335-Cells, Cultured,
pubmed-meshheading:11269335-Endothelium, Vascular,
pubmed-meshheading:11269335-Gene Transfer Techniques,
pubmed-meshheading:11269335-Genetic Vectors,
pubmed-meshheading:11269335-Humans,
pubmed-meshheading:11269335-Male,
pubmed-meshheading:11269335-Muscle, Smooth,
pubmed-meshheading:11269335-Rats,
pubmed-meshheading:11269335-Rats, Wistar,
pubmed-meshheading:11269335-Thrombin
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pubmed:articleTitle |
Increasing endothelial cell permeability improves the efficiency of myocyte adenoviral vector infection.
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pubmed:affiliation |
INSERM U460, Faculté de Médecine Xavier Bichat, Paris, France. nevo@bichat.inserm.fr
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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