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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2001-3-20
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pubmed:abstractText |
The failure of composite flaps despite anastomotic patency is thought to be mediated by the inflammatory response within the microvasculature, which results from unavoidable surgical trauma and transfer-related ischemia-reperfusion. Evidence suggests that stress conditioning may improve flap survival; however, the molecular mechanisms of protection are far from being clear. Therefore, we analyzed whether stress conditioning-induced heat-shock protein 32 is effective to prevent the inflammatory response in transferred osteomyocutaneous flaps.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0039-6060
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
129
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
292-301
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:11231457-Animals,
pubmed-meshheading:11231457-Cell Communication,
pubmed-meshheading:11231457-Conditioning (Psychology),
pubmed-meshheading:11231457-Endothelium, Vascular,
pubmed-meshheading:11231457-Heat-Shock Proteins,
pubmed-meshheading:11231457-Heme Oxygenase (Decyclizing),
pubmed-meshheading:11231457-Heme Oxygenase-1,
pubmed-meshheading:11231457-Hemodynamics,
pubmed-meshheading:11231457-Hot Temperature,
pubmed-meshheading:11231457-Inflammation,
pubmed-meshheading:11231457-Intercellular Adhesion Molecule-1,
pubmed-meshheading:11231457-Leukocytes,
pubmed-meshheading:11231457-Rats,
pubmed-meshheading:11231457-Rats, Wistar,
pubmed-meshheading:11231457-Stress, Physiological,
pubmed-meshheading:11231457-Surgical Flaps
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pubmed:year |
2001
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pubmed:articleTitle |
Reduction of inflammatory response in composite flap transfer by local stress conditioning-induced heat-shock protein 32.
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pubmed:affiliation |
Institute for Clinical and Experimental Surgery, Department of Oral and Maxillofacial Surgery, and the Clinic for Anesthesiology and Intensive Care Medicine, University of Saarland, Homburg/Saar, Germany.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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