pubmed-article:1119301 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:1119301 | lifeskim:mentions | umls-concept:C0019158 | lld:lifeskim |
pubmed-article:1119301 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:1119301 | lifeskim:mentions | umls-concept:C0023884 | lld:lifeskim |
pubmed-article:1119301 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
pubmed-article:1119301 | lifeskim:mentions | umls-concept:C2347379 | lld:lifeskim |
pubmed-article:1119301 | lifeskim:mentions | umls-concept:C0172367 | lld:lifeskim |
pubmed-article:1119301 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:1119301 | pubmed:dateCreated | 1975-5-29 | lld:pubmed |
pubmed-article:1119301 | pubmed:abstractText | Morphological investigations by lightmicroscopy were performed to evaluate the influence of an altered liver RES-function state on the course of Gal-N-hepatitis. There was a good correlation between the biochemical data and the morphological alterations. The simultaneous infusion of Gal-N 1 g/kg b.w. and carbon particles (2.6 mg/100 g b.w) into the portal vein was followed by subtotal necrosis of the liver and Sanarelli-Shwartzmann-phenomenon. Signs of RES-activity were nearly absent. 8 days after pretreatment with carbon, Gal-N application did not produce Gal-N hepatitis; 21 days after carbon infusion the morphological changes normally found after Gal-N application were present. The morphological studies confirm the concept that an altered RES-function state is a very important factor for the development of liver cell necrosis. | lld:pubmed |
pubmed-article:1119301 | pubmed:language | eng | lld:pubmed |
pubmed-article:1119301 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1119301 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:1119301 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1119301 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1119301 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:1119301 | pubmed:month | Feb | lld:pubmed |
pubmed-article:1119301 | pubmed:issn | 0300-970X | lld:pubmed |
pubmed-article:1119301 | pubmed:author | pubmed-author:LiehrHH | lld:pubmed |
pubmed-article:1119301 | pubmed:author | pubmed-author:GrünMM | lld:pubmed |
pubmed-article:1119301 | pubmed:author | pubmed-author:RasenackUU | lld:pubmed |
pubmed-article:1119301 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:1119301 | pubmed:volume | 22 | lld:pubmed |
pubmed-article:1119301 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:1119301 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:1119301 | pubmed:pagination | 29-35 | lld:pubmed |
pubmed-article:1119301 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:1119301 | pubmed:year | 1975 | lld:pubmed |
pubmed-article:1119301 | pubmed:articleTitle | Galactosamine- "hepatitis" in rats with different phagocytic activity of liver RES. | lld:pubmed |
pubmed-article:1119301 | pubmed:publicationType | Journal Article | lld:pubmed |