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rdf:type |
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lifeskim:mentions |
umls-concept:C0003009,
umls-concept:C0014442,
umls-concept:C0016169,
umls-concept:C0041904,
umls-concept:C0042333,
umls-concept:C0871261,
umls-concept:C1383860,
umls-concept:C1527148,
umls-concept:C1704632,
umls-concept:C1706817,
umls-concept:C2911692
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pubmed:issue |
7
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pubmed:dateCreated |
2001-2-22
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pubmed:abstractText |
Experimental and clinical evidence suggests that angiotensin II may be an important mediator of cardiac hypertrophy in response to hemodynamic stress. We investigated the effect of genetic variation in angiotensin-converting enzyme (ACE) on the development of cardiac hypertrophy and left ventricular (LV) dysfunction in response to volume overload.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
1524-4539
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:day |
20
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pubmed:volume |
103
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1012-6
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:11181478-Angiotensin II,
pubmed-meshheading:11181478-Animals,
pubmed-meshheading:11181478-Aorta,
pubmed-meshheading:11181478-Arteriovenous Fistula,
pubmed-meshheading:11181478-Cardiomegaly,
pubmed-meshheading:11181478-Chymases,
pubmed-meshheading:11181478-Disease Models, Animal,
pubmed-meshheading:11181478-Genetic Variation,
pubmed-meshheading:11181478-Male,
pubmed-meshheading:11181478-Mice,
pubmed-meshheading:11181478-Mice, Knockout,
pubmed-meshheading:11181478-Myocardium,
pubmed-meshheading:11181478-Organ Size,
pubmed-meshheading:11181478-Peptidyl-Dipeptidase A,
pubmed-meshheading:11181478-Renin-Angiotensin System,
pubmed-meshheading:11181478-Serine Endopeptidases,
pubmed-meshheading:11181478-Up-Regulation,
pubmed-meshheading:11181478-Vena Cava, Inferior,
pubmed-meshheading:11181478-Ventricular Dysfunction, Left
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pubmed:year |
2001
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pubmed:articleTitle |
Genetic variation in angiotensin-converting enzyme does not prevent development of cardiac hypertrophy or upregulation of angiotensin II in response to aortocaval fistula.
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pubmed:affiliation |
Birmingham VA Medical Center, University of Alabama at Birmingham, USA. gilbert.perry@med.va.gov
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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