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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2001-2-22
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pubmed:abstractText |
In in vivo models, radiation-induced genomic instability correlates with the risk of breast cancer development. In addition, homozygous mutations in tumor suppressor genes associated with breast cancer development adversely affects the processing and repair of radiation-induced DNA damage. We performed a case-control study to determine whether an assay measuring radiation-induced chromatid breaks correlated with the risk of having bilateral breast cancer.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0360-3016
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:day |
1
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pubmed:volume |
49
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
533-7
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:11173151-Adult,
pubmed-meshheading:11173151-Aged,
pubmed-meshheading:11173151-Breast Neoplasms,
pubmed-meshheading:11173151-Case-Control Studies,
pubmed-meshheading:11173151-Chromatids,
pubmed-meshheading:11173151-DNA Damage,
pubmed-meshheading:11173151-Female,
pubmed-meshheading:11173151-Genetic Predisposition to Disease,
pubmed-meshheading:11173151-Humans,
pubmed-meshheading:11173151-Lymphocytes,
pubmed-meshheading:11173151-Middle Aged,
pubmed-meshheading:11173151-Odds Ratio,
pubmed-meshheading:11173151-Phenotype,
pubmed-meshheading:11173151-Predictive Value of Tests,
pubmed-meshheading:11173151-Prospective Studies,
pubmed-meshheading:11173151-Radiation Tolerance
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pubmed:year |
2001
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pubmed:articleTitle |
Radiation-induced chromatid breaks as a predictor of breast cancer risk.
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pubmed:affiliation |
Department of Radiation Oncology, The University of Texas M. D. Anderson Cancer Center, Houston, TX 77030, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, U.S. Gov't, Non-P.H.S.
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