Linked Life Data

11122776

Source:http://linkedlifedata.com/resource/pubmed/id/11122776

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2001-1-26
pubmed:abstractText
Low levels of nitric oxide (NO) control the activities of guanylate cyclase and mitochondrial respiration. Increasing NO levels interact with multiple signaling systems through the formation of peroxynitrite and other oxidation products. Signaling mechanisms linked to NO participate in the prevention of acute responses such as vasoconstriction, thrombosis and the recruitment of inflammatory cells. In contrast, processes related to vascular remodeling, and responses to injury that are associated with the progression and adaptation to disease processes, are not as well understood. Many of the opposing processes involved in these adaptations may originate from the diverse signaling mechanisms that NO and its oxidized products can regulate in a cell-specific manner in the vessel wall.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Sep
pubmed:issn
1523-3804
pubmed:author
pubmed:issnType
Print
pubmed:volume
2
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
437-44
pubmed:dateRevised
2004-11-17
pubmed:meshHeading
pubmed:year
2000
pubmed:articleTitle
New concepts in vascular nitric oxide signaling.
pubmed:affiliation
Department of Physiology, New York Medical College, Valhalla, NY 10595, USA.
pubmed:publicationType
Journal Article, Review