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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2000-12-26
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pubmed:abstractText |
Loss of phospholipid asymmetry represents one of the hallmarks of apoptosis and results in the surface exposure of phosphatidylserine (PS) which can be indirectly monitored by the calcium-dependent binding of annexin V.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
1018-2438
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 2000 S. Karger AG, Basel
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pubmed:issnType |
Print
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pubmed:volume |
123
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
249-58
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pubmed:dateRevised |
2004-11-17
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pubmed:meshHeading |
pubmed-meshheading:11112862-Animals,
pubmed-meshheading:11112862-Apoptosis,
pubmed-meshheading:11112862-Cell Membrane,
pubmed-meshheading:11112862-Cells, Cultured,
pubmed-meshheading:11112862-Humans,
pubmed-meshheading:11112862-Male,
pubmed-meshheading:11112862-Mast Cells,
pubmed-meshheading:11112862-Mice,
pubmed-meshheading:11112862-Mice, Inbred BALB C,
pubmed-meshheading:11112862-Phosphatidylserines,
pubmed-meshheading:11112862-Rats,
pubmed-meshheading:11112862-Receptors, IgE
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pubmed:year |
2000
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pubmed:articleTitle |
Immunologic stimulation of mast cells leads to the reversible exposure of phosphatidylserine in the absence of apoptosis.
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pubmed:affiliation |
Unité Immuno-Allergie, Institut Pasteur, Paris, France.
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pubmed:publicationType |
Journal Article
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