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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2000-11-3
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pubmed:abstractText |
Abnormal accumulation of intracellular calcium following traumatic brain injury (TBI) is thought to contribute to a cascade of cellular events that lead to neuropathological conditions. Therefore, the possibility that specific calcium channel antagonists might exert neuroprotective effects in TBI has been of interest. The focus of this study was to examine whether Ziconotide produces such neuroprotective effects.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0022-3085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
93
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
821-8
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:11059664-Animals,
pubmed-meshheading:11059664-Behavior, Animal,
pubmed-meshheading:11059664-Brain Injuries,
pubmed-meshheading:11059664-Calcium Channel Blockers,
pubmed-meshheading:11059664-Disease Models, Animal,
pubmed-meshheading:11059664-Male,
pubmed-meshheading:11059664-Maze Learning,
pubmed-meshheading:11059664-Mitochondria,
pubmed-meshheading:11059664-Motor Activity,
pubmed-meshheading:11059664-Neuroprotective Agents,
pubmed-meshheading:11059664-Postural Balance,
pubmed-meshheading:11059664-Rats,
pubmed-meshheading:11059664-Rats, Sprague-Dawley,
pubmed-meshheading:11059664-omega-Conotoxins
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pubmed:year |
2000
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pubmed:articleTitle |
Neurobehavioral protection by the neuronal calcium channel blocker ziconotide in a model of traumatic diffuse brain injury in rats.
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pubmed:affiliation |
Department of Neurological Surgery, University of California at Davis Medical Center, Sacramento 95817, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
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