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| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C0965644 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C1333897 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C1704838 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C0679058 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C1547699 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C2700640 | lld:lifeskim |
| pubmed-article:11056102 | lifeskim:mentions | umls-concept:C0733688 | lld:lifeskim |
| pubmed-article:11056102 | pubmed:issue | 18 | lld:pubmed |
| pubmed-article:11056102 | pubmed:dateCreated | 2000-11-6 | lld:pubmed |
| pubmed-article:11056102 | pubmed:abstractText | Hypoxia-inducible factor-1 (HIF-1) is a heterodimeric transcription factor that regulates expression of genes involved in O(2) homeostasis, including vascular endothelial growth factor (VEGF), a potent stimulator of angiogenesis. We sought to exploit this native adaptive response to hypoxia as a treatment for chronic ischemia. | lld:pubmed |
| pubmed-article:11056102 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:11056102 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11056102 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11056102 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:11056102 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:GoldbergM AMA | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:IsnerJ MJM | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:SenS BSB | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:GregoryR JRJ | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:MagnerMM | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:AkitaG YGY | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:LuoYY | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:ShyuK GKG | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:JiangCC | lld:pubmed |
| pubmed-article:11056102 | pubmed:author | pubmed-author:VincentK AKA | lld:pubmed |
| pubmed-article:11056102 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:11056102 | pubmed:day | 31 | lld:pubmed |
| pubmed-article:11056102 | pubmed:volume | 102 | lld:pubmed |
| pubmed-article:11056102 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11056102 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11056102 | pubmed:pagination | 2255-61 | lld:pubmed |
| pubmed-article:11056102 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:11056102 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:11056102 | pubmed:articleTitle | Angiogenesis is induced in a rabbit model of hindlimb ischemia by naked DNA encoding an HIF-1alpha/VP16 hybrid transcription factor. | lld:pubmed |
| pubmed-article:11056102 | pubmed:affiliation | Genzyme Corp, Framingham, MA, USA. | lld:pubmed |
| pubmed-article:11056102 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11056102 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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