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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2000-11-27
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pubmed:abstractText |
Similarities in the hepatic responses to obesity and ethanol exposure suggest that these conditions evoke common pathogenic mechanisms. Thus, it is possible that ethanol exposure is increased in obesity. Given that intestinal bacteria can produce ethanol, the aim of this study was to determine if the intestinal production of ethanol is increased in obesity.
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pubmed:grant |
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0016-5085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
119
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1340-7
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:11054393-Administration, Oral,
pubmed-meshheading:11054393-Aging,
pubmed-meshheading:11054393-Animals,
pubmed-meshheading:11054393-Anti-Bacterial Agents,
pubmed-meshheading:11054393-Body Weight,
pubmed-meshheading:11054393-Breath Tests,
pubmed-meshheading:11054393-Circadian Rhythm,
pubmed-meshheading:11054393-Digestive System,
pubmed-meshheading:11054393-Ethanol,
pubmed-meshheading:11054393-Fatty Liver,
pubmed-meshheading:11054393-Male,
pubmed-meshheading:11054393-Mice,
pubmed-meshheading:11054393-Mice, Inbred C57BL,
pubmed-meshheading:11054393-Neomycin,
pubmed-meshheading:11054393-Obesity,
pubmed-meshheading:11054393-Reference Values
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pubmed:year |
2000
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pubmed:articleTitle |
Increased gastrointestinal ethanol production in obese mice: implications for fatty liver disease pathogenesis.
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pubmed:affiliation |
Departments of Environmental Health and Medicine, The Johns Hopkins University, Baltimore, Maryland 21205, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, U.S. Gov't, Non-P.H.S.
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