| pubmed-article:11041909 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C0029408 | lld:lifeskim |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C0389003 | lld:lifeskim |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C0521362 | lld:lifeskim |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C1257954 | lld:lifeskim |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C0762662 | lld:lifeskim |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C1707455 | lld:lifeskim |
| pubmed-article:11041909 | lifeskim:mentions | umls-concept:C2757001 | lld:lifeskim |
| pubmed-article:11041909 | pubmed:issue | 19 | lld:pubmed |
| pubmed-article:11041909 | pubmed:dateCreated | 2000-11-21 | lld:pubmed |
| pubmed-article:11041909 | pubmed:abstractText | Most nonsteroidal anti-inflammatory drugs (NSAIDs) are nonselective cyclooxygenase (COX-1 and COX-2) inhibitors and are associated with a variety of upper gastrointestinal (GI) tract symptoms. The roles of COX-1 and COX-2 in the pathogenesis of these symptoms are unclear. To test whether COX-2 inhibition with rofecoxib would have greater GI tolerability than nonselective COX-1 and COX-2 inhibition, we compared the incidences of (1) treatment discontinuations for GI adverse events (AEs) and (2) prespecified dyspeptic-type GI AEs among patients with osteoarthritis treated with rofecoxib vs NSAIDs. | lld:pubmed |
| pubmed-article:11041909 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11041909 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11041909 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:11041909 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11041909 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:11041909 | pubmed:issn | 0003-9926 | lld:pubmed |
| pubmed-article:11041909 | pubmed:author | pubmed-author:WatsonD JDJ | lld:pubmed |
| pubmed-article:11041909 | pubmed:author | pubmed-author:BologneseJ... | lld:pubmed |
| pubmed-article:11041909 | pubmed:author | pubmed-author:SimonT JTJ | lld:pubmed |
| pubmed-article:11041909 | pubmed:author | pubmed-author:QuanHH | lld:pubmed |
| pubmed-article:11041909 | pubmed:author | pubmed-author:HarperS ESE | lld:pubmed |
| pubmed-article:11041909 | pubmed:author | pubmed-author:ZhaoP LPL | lld:pubmed |
| pubmed-article:11041909 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11041909 | pubmed:day | 23 | lld:pubmed |
| pubmed-article:11041909 | pubmed:volume | 160 | lld:pubmed |
| pubmed-article:11041909 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11041909 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11041909 | pubmed:pagination | 2998-3003 | lld:pubmed |
| pubmed-article:11041909 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:11041909 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:11041909 | pubmed:articleTitle | Gastrointestinal tolerability of the selective cyclooxygenase-2 (COX-2) inhibitor rofecoxib compared with nonselective COX-1 and COX-2 inhibitors in osteoarthritis. | lld:pubmed |
| pubmed-article:11041909 | pubmed:affiliation | Merck Research Labs, West Point, PA, USA. | lld:pubmed |
| pubmed-article:11041909 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11041909 | pubmed:publicationType | Meta-Analysis | lld:pubmed |
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