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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2000-10-27
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pubmed:abstractText |
Neural mechanisms have been suggested to contribute to the pathogenesis of chronic asthma. The expression of neuropeptides such as substance P may be regulated by infectious pathogens, including Mycoplasma species. In contrast to substance P, the substance P receptor neurokinin 1 has not been examined at the protein level in asthmatic airways.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0091-6749
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
106
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
713-22
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:11031342-Adult,
pubmed-meshheading:11031342-Anti-Bacterial Agents,
pubmed-meshheading:11031342-Asthma,
pubmed-meshheading:11031342-Biopsy,
pubmed-meshheading:11031342-Bronchi,
pubmed-meshheading:11031342-Epithelium,
pubmed-meshheading:11031342-Female,
pubmed-meshheading:11031342-Forced Expiratory Volume,
pubmed-meshheading:11031342-Humans,
pubmed-meshheading:11031342-Male,
pubmed-meshheading:11031342-Mucus,
pubmed-meshheading:11031342-Mycoplasma pneumoniae,
pubmed-meshheading:11031342-Receptors, Neurokinin-1,
pubmed-meshheading:11031342-Substance P
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pubmed:year |
2000
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pubmed:articleTitle |
Substance P and its receptor neurokinin 1 expression in asthmatic airways.
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pubmed:affiliation |
Department of Medicine, National Jewish Medical and Research Center and the University of Colorado Health Sciences Center, Denver, CO 80206, USA.
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pubmed:publicationType |
Journal Article,
Clinical Trial,
Research Support, U.S. Gov't, P.H.S.,
Controlled Clinical Trial,
Research Support, Non-U.S. Gov't
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