| pubmed-article:11012762 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11012762 | lifeskim:mentions | umls-concept:C0015767 | lld:lifeskim |
| pubmed-article:11012762 | lifeskim:mentions | umls-concept:C0039195 | lld:lifeskim |
| pubmed-article:11012762 | lifeskim:mentions | umls-concept:C0020964 | lld:lifeskim |
| pubmed-article:11012762 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:11012762 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:11012762 | pubmed:dateCreated | 2000-10-16 | lld:pubmed |
| pubmed-article:11012762 | pubmed:abstractText | Feline leukaemia virus (FeLV) nucleic acid vaccination of domestic cats affords protection against viraemia and the development of latency without inducing antiviral antibodies.1 To determine the contribution of cell-mediated immunity to the control of virus replication and clearance from the host, FeLV-specific cytotoxic T lymphocyte (CTL) responses were compared in vaccine-protected, transiently viraemic, and persistently viraemic cats. Vaccinal immunity was associated with the detection of higher levels of virus-specific effector CTL in the peripheral blood and lymphoid organs to FeLV Gag/Pro and Env antigens than those observed in unvaccinated control, persistently viraemic cats (P<0.001). Likewise, higher levels of virus-specific CTLs were also observed in transiently viraemic cats which recovered following exposure to FeLV. In cats that controlled their infection, recognition of Gag/Pro antigens was significantly higher than the recognition of Env antigens. This is the first report highlighting the very significant role that virus-specific CTL have in determining the outcome of FeLV infection in either vaccinated cats or cats recovering naturally from FeLV exposure. | lld:pubmed |
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| pubmed-article:11012762 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11012762 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11012762 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11012762 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11012762 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:11012762 | pubmed:issn | 0019-2805 | lld:pubmed |
| pubmed-article:11012762 | pubmed:author | pubmed-author:JarrettOO | lld:pubmed |
| pubmed-article:11012762 | pubmed:author | pubmed-author:HanlonLL | lld:pubmed |
| pubmed-article:11012762 | pubmed:author | pubmed-author:FlynnJ NJN | lld:pubmed |
| pubmed-article:11012762 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11012762 | pubmed:volume | 101 | lld:pubmed |
| pubmed-article:11012762 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11012762 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11012762 | pubmed:pagination | 120-5 | lld:pubmed |
| pubmed-article:11012762 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:11012762 | pubmed:meshHeading | pubmed-meshheading:11012762... | lld:pubmed |
| pubmed-article:11012762 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:11012762 | pubmed:articleTitle | Feline leukaemia virus: protective immunity is mediated by virus-specific cytotoxic T lymphocytes. | lld:pubmed |
| pubmed-article:11012762 | pubmed:affiliation | Retrovirus Research Laboratory, Department of Veterinary Pathology, University of Glasgow, Glasgow, UK. | lld:pubmed |
| pubmed-article:11012762 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11012762 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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