| pubmed-article:10994752 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C0011923 | lld:lifeskim |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C1135918 | lld:lifeskim |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C0205042 | lld:lifeskim |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C2713359 | lld:lifeskim |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C1514893 | lld:lifeskim |
| pubmed-article:10994752 | lifeskim:mentions | umls-concept:C2699488 | lld:lifeskim |
| pubmed-article:10994752 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:10994752 | pubmed:dateCreated | 2000-12-28 | lld:pubmed |
| pubmed-article:10994752 | pubmed:abstractText | The observation of local 'elementary' Ca2+ release events (Ca2+ sparks) through ryanodine receptor (RyR) channels in the sarcoplasmic reticulum (SR) has changed our understanding of excitation-contraction (EC) coupling in cardiac and smooth muscle. In arterial smooth muscle, Ca2+ sparks have been suggested to oppose myogenic vasoconstriction and to influence vasorelaxation by activating co-localized Ca2+ activated K+ (K(Ca)) channels (STOCs). However, all prior studies on Ca2+ sparks have been performed in non-human tissues. | lld:pubmed |
| pubmed-article:10994752 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10994752 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10994752 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10994752 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10994752 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:10994752 | pubmed:issn | 0263-6352 | lld:pubmed |
| pubmed-article:10994752 | pubmed:author | pubmed-author:LuftF CFC | lld:pubmed |
| pubmed-article:10994752 | pubmed:author | pubmed-author:HallerHH | lld:pubmed |
| pubmed-article:10994752 | pubmed:author | pubmed-author:FürstenauMM | lld:pubmed |
| pubmed-article:10994752 | pubmed:author | pubmed-author:RielKK | lld:pubmed |
| pubmed-article:10994752 | pubmed:author | pubmed-author:GollaschMM | lld:pubmed |
| pubmed-article:10994752 | pubmed:author | pubmed-author:LöhnMM | lld:pubmed |
| pubmed-article:10994752 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10994752 | pubmed:volume | 18 | lld:pubmed |
| pubmed-article:10994752 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10994752 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10994752 | pubmed:pagination | 1215-22 | lld:pubmed |
| pubmed-article:10994752 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:10994752 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10994752 | pubmed:articleTitle | Calcium sparks in human coronary artery smooth muscle cells resolved by confocal imaging. | lld:pubmed |
| pubmed-article:10994752 | pubmed:affiliation | Franz Volhard Clinic at the Max Delbrück Center for Molecular Medicine, Charité University Hospitals, Humboldt University Berlin, Germany. | lld:pubmed |
| pubmed-article:10994752 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10994752 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:10994752 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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