| pubmed-article:10992518 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C0445750 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C0085732 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C0596988 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C1707455 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C0449943 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C1551336 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C1548795 | lld:lifeskim |
| pubmed-article:10992518 | lifeskim:mentions | umls-concept:C0442335 | lld:lifeskim |
| pubmed-article:10992518 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:10992518 | pubmed:dateCreated | 2000-11-3 | lld:pubmed |
| pubmed-article:10992518 | pubmed:abstractText | We compared the ability of Salmonella enterica serovar Typhimurium SL1344 aroA aroD (BRD509) and aroA htrA (BRD807) mutants to act as live vectors for delivery of fragment C of tetanus toxin (FrgC). FrgC was expressed in these strains from either pTETnir15 or pTEThtrA1. BRD509FrgC(+) strains elicited approximately 2-log-higher serum anti-FrgC antibody titers than BRD807FrgC(+) strains. All mice immunized with BRD807pTEThtrA1, BRD509pTEThtrA1, and BRD509pTETnir15 (but not BRD807pTETnir15) were protected against tetanus. | lld:pubmed |
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| pubmed-article:10992518 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:10992518 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10992518 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10992518 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:10992518 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:10992518 | pubmed:author | pubmed-author:RobertsMM | lld:pubmed |
| pubmed-article:10992518 | pubmed:author | pubmed-author:OOIS KSK | lld:pubmed |
| pubmed-article:10992518 | pubmed:author | pubmed-author:PickardDD | lld:pubmed |
| pubmed-article:10992518 | pubmed:author | pubmed-author:ChatfieldSS | lld:pubmed |
| pubmed-article:10992518 | pubmed:author | pubmed-author:BacosJJ | lld:pubmed |
| pubmed-article:10992518 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10992518 | pubmed:volume | 68 | lld:pubmed |
| pubmed-article:10992518 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10992518 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10992518 | pubmed:pagination | 6041-3 | lld:pubmed |
| pubmed-article:10992518 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:10992518 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10992518 | pubmed:articleTitle | Comparison of abilities of Salmonella enterica serovar typhimurium aroA aroD and aroA htrA mutants to act as live vectors. | lld:pubmed |
| pubmed-article:10992518 | pubmed:affiliation | Department of Veterinary Pathology, Glasgow University Veterinary School, Glasgow G61 1QH, United Kingdom. M.Roberts@vet.gla.ac.uk | lld:pubmed |
| pubmed-article:10992518 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10992518 | pubmed:publicationType | Comparative Study | lld:pubmed |
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