| pubmed-article:10869259 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0026473 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0205042 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0001511 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0128897 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C0041904 | lld:lifeskim |
| pubmed-article:10869259 | lifeskim:mentions | umls-concept:C1417947 | lld:lifeskim |
| pubmed-article:10869259 | pubmed:issue | 25 | lld:pubmed |
| pubmed-article:10869259 | pubmed:dateCreated | 2000-7-19 | lld:pubmed |
| pubmed-article:10869259 | pubmed:abstractText | We have recently demonstrated a lectin-like receptor for oxidized (ox)-LDL (LOX-1) in human coronary artery endothelial cells (HCAECs). This receptor is upregulated by ox-LDL. The present study examined the significance of LOX-1 in monocyte adhesion to HCAECs and endothelial injury in response to ox-LDL. | lld:pubmed |
| pubmed-article:10869259 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10869259 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10869259 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:10869259 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10869259 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:10869259 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:10869259 | pubmed:author | pubmed-author:LUPP | lld:pubmed |
| pubmed-article:10869259 | pubmed:author | pubmed-author:MehtaJ LJL | lld:pubmed |
| pubmed-article:10869259 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:10869259 | pubmed:day | 27 | lld:pubmed |
| pubmed-article:10869259 | pubmed:volume | 101 | lld:pubmed |
| pubmed-article:10869259 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10869259 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10869259 | pubmed:pagination | 2889-95 | lld:pubmed |
| pubmed-article:10869259 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:10869259 | pubmed:meshHeading | pubmed-meshheading:10869259... | lld:pubmed |
| pubmed-article:10869259 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10869259 | pubmed:articleTitle | Antisense to LOX-1 inhibits oxidized LDL-mediated upregulation of monocyte chemoattractant protein-1 and monocyte adhesion to human coronary artery endothelial cells. | lld:pubmed |
| pubmed-article:10869259 | pubmed:affiliation | Departments of Medicine and Physiology, University of Florida and VA Medical Center, Gainesville, FL 32610, USA. | lld:pubmed |
| pubmed-article:10869259 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10869259 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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