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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2000-6-21
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pubmed:abstractText |
Cystic fibrosis transmembrane conductance regulator (CFTR) Cl(-) channels play an important role in HCO(3)(-) secretion by pancreatic duct cells (PDCs). Our aims were to characterize the CFTR conductance of guinea pig PDCs and to establish whether CFTR is regulated by HCO(3)(-).
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
0016-5085
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
118
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1187-96
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:10833494-Animals,
pubmed-meshheading:10833494-Anions,
pubmed-meshheading:10833494-Bicarbonates,
pubmed-meshheading:10833494-Carbon Dioxide,
pubmed-meshheading:10833494-Chloride Channels,
pubmed-meshheading:10833494-Chlorides,
pubmed-meshheading:10833494-Cyclic AMP,
pubmed-meshheading:10833494-Cystic Fibrosis,
pubmed-meshheading:10833494-Cystic Fibrosis Transmembrane Conductance Regulator,
pubmed-meshheading:10833494-Electric Conductivity,
pubmed-meshheading:10833494-Electrophysiology,
pubmed-meshheading:10833494-Female,
pubmed-meshheading:10833494-Forskolin,
pubmed-meshheading:10833494-Guinea Pigs,
pubmed-meshheading:10833494-Hydrogen-Ion Concentration,
pubmed-meshheading:10833494-Male,
pubmed-meshheading:10833494-Membrane Potentials,
pubmed-meshheading:10833494-Pancreatic Ducts,
pubmed-meshheading:10833494-Pancreatic Juice,
pubmed-meshheading:10833494-Secretin,
pubmed-meshheading:10833494-Sodium Bicarbonate
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pubmed:year |
2000
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pubmed:articleTitle |
Cystic fibrosis transmembrane conductance regulator currents in guinea pig pancreatic duct cells: inhibition by bicarbonate ions.
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pubmed:affiliation |
Department of Physiological Sciences, University Medical School, Newcastle upon Tyne, England.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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