Source:http://linkedlifedata.com/resource/pubmed/id/10801751
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
18
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pubmed:dateCreated |
2000-5-30
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pubmed:abstractText |
Calcineurin may play a pivotal role in the signaling of cardiac hypertrophy; since this hypothesis was first put forward, controversial reports have been published using various experimental models. This study was designed to compare the physiological left ventricular hypertrophy (LVH) induced by voluntary exercise with LVH induced by aortic constriction and to determine whether calcineurin participates in the signaling of exercise-induced LVH.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1524-4539
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pubmed:author | |
pubmed:issnType |
Electronic
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pubmed:day |
9
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pubmed:volume |
101
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2134-7
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading | |
pubmed:year |
2000
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pubmed:articleTitle |
Calcineurin is activated in rat hearts with physiological left ventricular hypertrophy induced by voluntary exercise training.
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pubmed:affiliation |
Department of Cardiovascular Medicine, University of Tokyo, Japan.
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pubmed:publicationType |
Journal Article
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