Source:http://linkedlifedata.com/resource/pubmed/id/10782912
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4
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pubmed:dateCreated |
2000-7-31
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pubmed:abstractText |
It has previously been shown in rat liver that the gap junctional proteins connexin32 and connexin26 are downregulated when murine hepatocytes are in the S-phase of the cell cycle. Therefore, it has been hypothesized that loss of functional gap junctions could affect proliferation of hepatocytes. This study aimed to check this hypothesis.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0168-8278
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
32
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
627-35
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:10782912-Animals,
pubmed-meshheading:10782912-Connexins,
pubmed-meshheading:10782912-DNA,
pubmed-meshheading:10782912-Gap Junctions,
pubmed-meshheading:10782912-Gene Expression Regulation,
pubmed-meshheading:10782912-Liver Regeneration,
pubmed-meshheading:10782912-Mice,
pubmed-meshheading:10782912-Mice, Knockout,
pubmed-meshheading:10782912-Rats
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pubmed:year |
2000
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pubmed:articleTitle |
The extent of synchronous initiation and termination of DNA synthesis in regenerating mouse liver is dependent on connexin32 expressing gap junctions.
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pubmed:affiliation |
Institut für Genetik, Abt. Molekulargenetik, University of Bonn, Germany.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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