10757217

Source:http://linkedlifedata.com/resource/pubmed/id/10757217

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0022661, umls-concept:C0035820, umls-concept:C0175677
pubmed:issue	2
pubmed:dateCreated	2000-5-25
pubmed:abstractText	Progressive renal failure results from a triad of glomerulosclerosis, tubulointerstitial fibrosis and vascular sclerosis. The mechanisms by which tubules are injured, and by which the tubular epithelial cell then excites interstitial inflammation culminating in fibroblast activation and fibrosis have become increasingly understood. Most current methods to prevent progressive glomerulosclerosis would inherently prevent tubular injury and interstitial fibrosis. The behaviour and control of the renal fibroblast is being investigated, with the potential for direct interference with its functions.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9303753
pubmed:citationSubset	IM
pubmed:status	MEDLINE
pubmed:month	Mar
pubmed:issn	1062-4821
pubmed:author	pubmed-author:BeckerG JGJ, pubmed-author:HewitsonT DTD
pubmed:issnType	Print
pubmed:volume	9
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	133-8
pubmed:dateRevised	2006-11-15
pubmed:meshHeading	pubmed-meshheading:10757217-Disease Progression, pubmed-meshheading:10757217-Fibroblasts, pubmed-meshheading:10757217-Fibrosis, pubmed-meshheading:10757217-Humans, pubmed-meshheading:10757217-Kidney Failure, Chronic, pubmed-meshheading:10757217-Kidney Tubules
pubmed:year	2000
pubmed:articleTitle	The role of tubulointerstitial injury in chronic renal failure.
pubmed:affiliation	Department of Nephrology, The Royal Melbourne Hospital, Parkville, Victoria, Australia. gavin.becker@nwhcn.org.au
pubmed:publicationType	Journal Article, Review, Research Support, Non-U.S. Gov't