pubmed-article:10698741 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10698741 | lifeskim:mentions | umls-concept:C0036111 | lld:lifeskim |
pubmed-article:10698741 | lifeskim:mentions | umls-concept:C0031307 | lld:lifeskim |
pubmed-article:10698741 | lifeskim:mentions | umls-concept:C0068355 | lld:lifeskim |
pubmed-article:10698741 | lifeskim:mentions | umls-concept:C1257893 | lld:lifeskim |
pubmed-article:10698741 | pubmed:issue | 5458 | lld:pubmed |
pubmed-article:10698741 | pubmed:dateCreated | 2000-3-23 | lld:pubmed |
pubmed-article:10698741 | pubmed:abstractText | A type III protein secretion system encoded by Salmonella pathogenicity island 2 (SPI2) has been found to be required for virulence and survival within macrophages. Here, SPI2 was shown to allow Salmonella typhimurium to avoid NADPH oxidase-dependent killing by macrophages. The ability of SPI2-mutant bacteria to survive in macrophages and to cause lethal infection in mice was restored by abrogation of the NADPH oxidase-dependent respiratory burst. Ultrastructural and immunofluorescence microscopy demonstrated efficient localization of the NADPH oxidase in the proximity of vacuoles containing SPI2-mutant but not wild-type bacteria, suggesting that SPI2 interferes with trafficking of oxidase-containing vesicles to the phagosome. | lld:pubmed |
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pubmed-article:10698741 | pubmed:language | eng | lld:pubmed |
pubmed-article:10698741 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10698741 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:10698741 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10698741 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10698741 | pubmed:month | Mar | lld:pubmed |
pubmed-article:10698741 | pubmed:issn | 0036-8075 | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:MastroeniPP | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:XuYY | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:DinauerM CMC | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:HoldenD WDW | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:FangF CFC | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:Vazquez-Torre... | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:Jones-CarsonJ... | lld:pubmed |
pubmed-article:10698741 | pubmed:author | pubmed-author:LuciaS MSM | lld:pubmed |
pubmed-article:10698741 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10698741 | pubmed:day | 3 | lld:pubmed |
pubmed-article:10698741 | pubmed:volume | 287 | lld:pubmed |
pubmed-article:10698741 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10698741 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10698741 | pubmed:pagination | 1655-8 | lld:pubmed |
pubmed-article:10698741 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:10698741 | pubmed:year | 2000 | lld:pubmed |
pubmed-article:10698741 | pubmed:articleTitle | Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase. | lld:pubmed |
pubmed-article:10698741 | pubmed:affiliation | Department of Medicine, University of Colorado Health Sciences Center, Denver, CO 80262, USA. | lld:pubmed |
pubmed-article:10698741 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10698741 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:10698741 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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