| pubmed-article:10652030 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0034493 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0034705 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0013030 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0232105 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0597484 | lld:lifeskim |
| pubmed-article:10652030 | lifeskim:mentions | umls-concept:C0598850 | lld:lifeskim |
| pubmed-article:10652030 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:10652030 | pubmed:dateCreated | 2000-3-9 | lld:pubmed |
| pubmed-article:10652030 | pubmed:abstractText | Dopamine (DA) is thought to regulate renal proximal transport through the inhibition of the Na+,K+-ATPase and/or Na+/H+ exchanger. Defects in this dopaminergic system are proposed to be a pathogenic factor of genetic hypertension. However, microperfusion studies have not consistently confirmed direct tubular effects of DA. | lld:pubmed |
| pubmed-article:10652030 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10652030 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10652030 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10652030 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10652030 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10652030 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10652030 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:10652030 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:KimuraSS | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:FujitaTT | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:GotoAA | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:TaniguchiSS | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:HartPP | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:UwatokoSS | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:SekiGG | lld:pubmed |
| pubmed-article:10652030 | pubmed:author | pubmed-author:KunimiMM | lld:pubmed |
| pubmed-article:10652030 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10652030 | pubmed:volume | 57 | lld:pubmed |
| pubmed-article:10652030 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10652030 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10652030 | pubmed:pagination | 534-43 | lld:pubmed |
| pubmed-article:10652030 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:10652030 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10652030 | pubmed:articleTitle | Dopamine inhibits renal Na+:HCO3- cotransporter in rabbits and normotensive rats but not in spontaneously hypertensive rats. | lld:pubmed |
| pubmed-article:10652030 | pubmed:affiliation | Department of Nephrology and Endocrinology, and Department of Infectious Diseases, Faculty of Medicine, University of Tokyo, Japan. | lld:pubmed |
| pubmed-article:10652030 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10652030 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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