rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
3
|
pubmed:dateCreated |
2000-2-10
|
pubmed:abstractText |
Dihydropyridine calcium channel blockers protect endothelial cells against ischemia and reperfusion injury, suggesting that nifedipine may increase the in vivo cardiac NO level and thus coronary blood flow (CBF) in ischemic hearts. We tested this hypothesis.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Jan
|
pubmed:issn |
1524-4539
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:day |
25
|
pubmed:volume |
101
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
311-7
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:10645928-Animals,
pubmed-meshheading:10645928-Bradykinin,
pubmed-meshheading:10645928-Calcium Channel Blockers,
pubmed-meshheading:10645928-Coronary Vessels,
pubmed-meshheading:10645928-Cyclic GMP,
pubmed-meshheading:10645928-Dogs,
pubmed-meshheading:10645928-Heart Rate,
pubmed-meshheading:10645928-Myocardial Ischemia,
pubmed-meshheading:10645928-Nifedipine,
pubmed-meshheading:10645928-Nitric Oxide,
pubmed-meshheading:10645928-Systole,
pubmed-meshheading:10645928-Vasodilation
|
pubmed:year |
2000
|
pubmed:articleTitle |
Nifedipine-induced coronary vasodilation in ischemic hearts is attributable to bradykinin- and NO-dependent mechanisms in dogs.
|
pubmed:affiliation |
Department of Internal Medicine, Osaka University Graduate School of Medicine, Suita, Japan. kitakaze@medone.med.osaka-u.ac.jp
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|