Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7
pubmed:dateCreated
2000-2-8
pubmed:abstractText
Ischemia reperfusion injury is an uncommon kind of inflammation. Oxygen deprivation is followed by cellular dysfunction, reversible or not and mainly tubular. Ischemia initiates and amplifies the fundamental events of reperfusion injury, that are going to occur after oxygen reintroduction. Reperfusion is characterized by the production of oxygen reactive species, endothelial activation, endothelial release of chemoattractants, chemokines and cytokines, expression of adhesion molecules, that are going to mediate the recruitment and activation of circulating leucocytes, later responsible for the main lesions of reperfusion injury. Studies in kidney transplantation have brought evidence that the fibrotic healing of reperfusion injury have long term and significant consequences on kidney function.
pubmed:language
fre
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
0250-4960
pubmed:author
pubmed:issnType
Print
pubmed:volume
20
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
371-5
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed:year
1999
pubmed:articleTitle
[Renal ischemia reperfusion syndrome].
pubmed:affiliation
Service de néphrologie et d'immunologie clinique. Hôtel Dieu, CHU, Nantes.
pubmed:publicationType
Journal Article, English Abstract, Review