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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2000-1-3
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pubmed:abstractText |
Nitric oxide (NO) participates in the regulation of hemodynamic and microcirculatory changes in intestinal ischemia and reperfusion (I/R). However, the nature of the involvement of an inducible NO release has been controversial. This study evaluates the impact of an inducible NO synthase inhibitor, aminoguanidine, used as a treatment in a rat intestinal I/R model.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0022-5282
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
47
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1108-13
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pubmed:dateRevised |
2010-1-15
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pubmed:meshHeading |
pubmed-meshheading:10608542-Animals,
pubmed-meshheading:10608542-Blood Flow Velocity,
pubmed-meshheading:10608542-Disease Models, Animal,
pubmed-meshheading:10608542-Drug Evaluation, Preclinical,
pubmed-meshheading:10608542-Granulocytes,
pubmed-meshheading:10608542-Guanidines,
pubmed-meshheading:10608542-Hemodynamics,
pubmed-meshheading:10608542-Humans,
pubmed-meshheading:10608542-Intestines,
pubmed-meshheading:10608542-Laser-Doppler Flowmetry,
pubmed-meshheading:10608542-Lipid Peroxidation,
pubmed-meshheading:10608542-Liver,
pubmed-meshheading:10608542-Macrophage-1 Antigen,
pubmed-meshheading:10608542-Microcirculation,
pubmed-meshheading:10608542-Nitric Oxide Synthase,
pubmed-meshheading:10608542-Rats,
pubmed-meshheading:10608542-Rats, Sprague-Dawley,
pubmed-meshheading:10608542-Reperfusion Injury,
pubmed-meshheading:10608542-Time Factors,
pubmed-meshheading:10608542-Venules
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pubmed:year |
1999
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pubmed:articleTitle |
Aminoguanidine attenuates hemodynamic and microcirculatory derangement in rat intestinal ischemia and reperfusion.
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pubmed:affiliation |
Department of Surgery, Chang Gung Memorial Hospital, Chang Gung University College of Medicine, Taipei, Taiwan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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