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rdf:type |
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lifeskim:mentions |
umls-concept:C0002844,
umls-concept:C0085862,
umls-concept:C0334227,
umls-concept:C0376358,
umls-concept:C0871261,
umls-concept:C1325288,
umls-concept:C1527148,
umls-concept:C1704632,
umls-concept:C1706817,
umls-concept:C2825032,
umls-concept:C2911692
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pubmed:issue |
21
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pubmed:dateCreated |
1999-11-30
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pubmed:abstractText |
Androgen withdrawal is a standard therapy for prostate cancer that results in a decrease in tumor volume and a decline in serum prostate-specific antigen in the majority of patients. To understand the factors associated with regression of prostate cancers after androgen withdrawal, we studied cell cycle regulator changes in the CWR22 human prostate cancer xenograft model.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Androgens,
http://linkedlifedata.com/resource/pubmed/chemical/Antibodies, Monoclonal,
http://linkedlifedata.com/resource/pubmed/chemical/CDKN1A protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Cdkn1a protein, mouse,
http://linkedlifedata.com/resource/pubmed/chemical/Cyclin-Dependent Kinase Inhibitor...,
http://linkedlifedata.com/resource/pubmed/chemical/Cyclin-Dependent Kinase Inhibitor...,
http://linkedlifedata.com/resource/pubmed/chemical/Cyclins,
http://linkedlifedata.com/resource/pubmed/chemical/Ki-67 Antigen,
http://linkedlifedata.com/resource/pubmed/chemical/Microfilament Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Muscle Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Androgen,
http://linkedlifedata.com/resource/pubmed/chemical/Tagln protein, mouse,
http://linkedlifedata.com/resource/pubmed/chemical/Tumor Suppressor Protein p53
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0027-8874
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:day |
3
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pubmed:volume |
91
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1869-76
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:10547394-Androgens,
pubmed-meshheading:10547394-Animals,
pubmed-meshheading:10547394-Antibodies, Monoclonal,
pubmed-meshheading:10547394-Cell Cycle,
pubmed-meshheading:10547394-Cyclin-Dependent Kinase Inhibitor p16,
pubmed-meshheading:10547394-Cyclin-Dependent Kinase Inhibitor p21,
pubmed-meshheading:10547394-Cyclins,
pubmed-meshheading:10547394-Disease Models, Animal,
pubmed-meshheading:10547394-Gene Expression Regulation, Neoplastic,
pubmed-meshheading:10547394-Humans,
pubmed-meshheading:10547394-Immunohistochemistry,
pubmed-meshheading:10547394-Ki-67 Antigen,
pubmed-meshheading:10547394-Male,
pubmed-meshheading:10547394-Mice,
pubmed-meshheading:10547394-Mice, Inbred BALB C,
pubmed-meshheading:10547394-Mice, Nude,
pubmed-meshheading:10547394-Microfilament Proteins,
pubmed-meshheading:10547394-Muscle Proteins,
pubmed-meshheading:10547394-Prostatic Neoplasms,
pubmed-meshheading:10547394-Receptors, Androgen,
pubmed-meshheading:10547394-Time Factors,
pubmed-meshheading:10547394-Transplantation, Heterologous,
pubmed-meshheading:10547394-Tumor Suppressor Protein p53
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pubmed:year |
1999
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pubmed:articleTitle |
Prostate cancer cell cycle regulators: response to androgen withdrawal and development of androgen independence.
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pubmed:affiliation |
Department of Medicine, Sloan-Kettering Institute, Memorial Sloan-Kettering Cancer Center, New York, NY, USA. d-agus@ski.mskcc.org
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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