| pubmed-article:10445029 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10445029 | lifeskim:mentions | umls-concept:C0043393 | lld:lifeskim |
| pubmed-article:10445029 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:10445029 | lifeskim:mentions | umls-concept:C1167093 | lld:lifeskim |
| pubmed-article:10445029 | lifeskim:mentions | umls-concept:C1336789 | lld:lifeskim |
| pubmed-article:10445029 | lifeskim:mentions | umls-concept:C0599894 | lld:lifeskim |
| pubmed-article:10445029 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:10445029 | pubmed:dateCreated | 1999-8-30 | lld:pubmed |
| pubmed-article:10445029 | pubmed:abstractText | Genetic and biochemical studies indicate that the evolutionarily conserved Swi/Snf complex acts at a subset of genes to help transcriptional activators function on chromatin templates. The mechanism by which this complex is targeted to specific chromosomal loci remains unknown. We show that Swi/Snf is required for expression of the yeast histone HTA1-HTB1 locus because of the role of Hir1p and Hir2p corepressors in negatively regulating transcription. Snf5p, Snf2p/Swi2p, and Swi3p, three components of the yeast Swi/Snf complex, coimmunoprecipitate with each Hir protein, and Snf5p is maximally associated with the HTA1-HTB1 promoter when the Hir-based repression system is intact and the Swi/Snf complex is functional. The data support a role for the Hir repressors in the gene-specific targeting of Swi/Snf. | lld:pubmed |
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| pubmed-article:10445029 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:10445029 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10445029 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10445029 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:10445029 | pubmed:issn | 1097-2765 | lld:pubmed |
| pubmed-article:10445029 | pubmed:author | pubmed-author:EguchiSS | lld:pubmed |
| pubmed-article:10445029 | pubmed:author | pubmed-author:OsleyM AMA | lld:pubmed |
| pubmed-article:10445029 | pubmed:author | pubmed-author:NackerdienZZ | lld:pubmed |
| pubmed-article:10445029 | pubmed:author | pubmed-author:DimovaDD | lld:pubmed |
| pubmed-article:10445029 | pubmed:author | pubmed-author:FurgesonSS | lld:pubmed |
| pubmed-article:10445029 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10445029 | pubmed:volume | 4 | lld:pubmed |
| pubmed-article:10445029 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10445029 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10445029 | pubmed:pagination | 75-83 | lld:pubmed |
| pubmed-article:10445029 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:10445029 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10445029 | pubmed:articleTitle | A role for transcriptional repressors in targeting the yeast Swi/Snf complex. | lld:pubmed |
| pubmed-article:10445029 | pubmed:affiliation | Program in Molecular Biology, Sloan Kettering Cancer Center, New York, New York 10021, USA. | lld:pubmed |
| pubmed-article:10445029 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10445029 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:10445029 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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