Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
7
pubmed:dateCreated
1999-9-13
pubmed:abstractText
Maize leaves have a stereotypical pattern of cell types organized into discrete domains. These domains are altered by mutations in knotted1 (kn1) and knox (for kn1-like homeobox) genes. Gnarley (Gn1) is a dominant maize mutant that exhibits many of the phenotypic characteristics of the kn1 family of mutants. Gn1 is unique because it changes parameters of cell growth in the basal-most region of the leaf, the sheath, resulting in dramatically altered sheath morphology. The strongly expressive allele Gn1-R also gives rise to a floral phenotype in which ectopic carpels form. Introgression studies showed that the severity of the Gn1-conferred phenotype is strongly influenced by genetic background. Gn1 maps to knox4, and knox4 is ectopically expressed in plants with the Gn1-conferred phenotype. Immunolocalization experiments showed that the KNOX protein accumulates at the base of Gn1 leaves in a pattern that is spatially and temporally correlated with appearance of the mutant phenotype. We further demonstrate that Gn1 is knox4 by correlating loss of the mutant phenotype with insertion of a Mutator transposon into knox4.
pubmed:commentsCorrections
http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-10022841, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-11038553, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-1313711, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-1333722, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-1362381, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-16453866, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-1672445, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-17246303, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-17246413, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-2088734, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-2391003, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-7557382, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-7684007, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-7715728, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-7734963, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-7866030, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-7896105, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8103379, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8103599, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8138166, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8538741, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8625799, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8625845, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8631155, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8674408, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8970728, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-8978070, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9254931, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9272946, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9286690, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9362467, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9367429, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9437860, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9655808, http://linkedlifedata.com/resource/pubmed/commentcorrection/10402426-9847244
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
1040-4651
pubmed:author
pubmed:issnType
Print
pubmed:volume
11
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1239-52
pubmed:dateRevised
2010-9-13
pubmed:meshHeading
pubmed:year
1999
pubmed:articleTitle
Gnarley1 is a dominant mutation in the knox4 homeobox gene affecting cell shape and identity.
pubmed:affiliation
Department of Plant and Microbial Biology, University of California, Berkeley, California 94720, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, Non-P.H.S., Research Support, Non-U.S. Gov't