10355299

Source:http://linkedlifedata.com/resource/pubmed/id/10355299

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0025936, umls-concept:C0034790, umls-concept:C0162638, umls-concept:C1336745, umls-concept:C1704259, umls-concept:C1705987, umls-concept:C1710082, umls-concept:C2699787
pubmed:issue	2
pubmed:dateCreated	1999-8-3
pubmed:abstractText	The abrogation of apoptosis during development of thymocytes could contribute to the lymphomagenesis of immature thymocytes which otherwise would be deleted. In agreement with this possibility it was found that cells of thymic lymphomas from TCR anty H-Y transgenic mice were resistant to TCR mediated apoptosis. Molecular mechanism responsible for resistance to TCR/Nur77-mediated apoptosis in these lymphomas is discussed.
pubmed:language	pol
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0421052
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Antigen, T-Cell
pubmed:status	MEDLINE
pubmed:issn	0032-5449
pubmed:author	pubmed-author:Strzada?aLL
pubmed:issnType	Print
pubmed:volume	53
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	345-50
pubmed:dateRevised	2006-11-15
pubmed:meshHeading	pubmed-meshheading:10355299-Animals, pubmed-meshheading:10355299-Apoptosis, pubmed-meshheading:10355299-Lymphoma, pubmed-meshheading:10355299-Mice, pubmed-meshheading:10355299-Mice, Transgenic, pubmed-meshheading:10355299-Receptors, Antigen, T-Cell, pubmed-meshheading:10355299-Signal Transduction, pubmed-meshheading:10355299-Thymus Neoplasms
pubmed:year	1999
pubmed:articleTitle	[Disturbance of signalling pathways for apoptosis in thymic lymphomas from TCR transgenic mice].
pubmed:affiliation	Laboratorium Immunologii Komórkowej, Instytutu Immunologii i Terapii Do?wiadczalnej PAN, Wroc?awiu.
pubmed:publicationType	Journal Article, English Abstract, Review