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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
1999-7-6
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pubmed:abstractText |
Interferon alpha-2b (IFNalpha) treatment of diseases can be accompanied by impaired renal function and capillary leak syndrome. To explore potential mechanisms of IFNalpha-induced renal dysfunction, an in vitro cell culture model system was established to investigate the effects of IFNalpha on barrier function and junctional complexes.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
0085-2538
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
55
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2178-91
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pubmed:dateRevised |
2011-11-17
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pubmed:meshHeading |
pubmed-meshheading:10354267-Animals,
pubmed-meshheading:10354267-Cadherins,
pubmed-meshheading:10354267-Cell Membrane Permeability,
pubmed-meshheading:10354267-Electric Impedance,
pubmed-meshheading:10354267-Interferon-alpha,
pubmed-meshheading:10354267-Kidney Tubules, Proximal,
pubmed-meshheading:10354267-LLC-PK1 Cells,
pubmed-meshheading:10354267-Membrane Proteins,
pubmed-meshheading:10354267-Microscopy, Electron,
pubmed-meshheading:10354267-Microscopy, Fluorescence,
pubmed-meshheading:10354267-Phosphoproteins,
pubmed-meshheading:10354267-Phosphorylation,
pubmed-meshheading:10354267-Recombinant Proteins,
pubmed-meshheading:10354267-Swine,
pubmed-meshheading:10354267-Tight Junctions,
pubmed-meshheading:10354267-Tyrosine
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pubmed:year |
1999
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pubmed:articleTitle |
Effects of interferon alpha-2b on barrier function and junctional complexes of renal proximal tubular LLC-PK1 cells.
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pubmed:affiliation |
Institute of Physiology, University of Innsburck, Austria.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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