Source:http://linkedlifedata.com/resource/pubmed/id/10335863
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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
2
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| pubmed:dateCreated |
1999-8-19
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| pubmed:abstractText |
The etiology of sporadic acute hepatitis was studied in 334 consecutive patients from Taiwan (237 men and 97 women, aged 16-81 years), with emphasis on the role of hepatitis C virus (HCV), hepatitis E virus (HEV), and GB virus-C/hepatitis G virus (GBV-C/HGV) in acute non-A, non-B (NANB) hepatitis and in HBsAg carriers with superimposed acute hepatitis. According to the conventional diagnostic criteria, there were 12 cases (3.6%) of acute hepatitis A, 17 cases (5.1%) of acute hepatitis B, 128 cases (38.3%) of acute NANB hepatitis, and 177 cases (53.0%) of acute hepatitis in HBsAg carriers (those who were HBsAg positive but IgM anti-HBc negative). Among 128 cases of acute NANB hepatitis, 70 (54.7%) had acute hepatitis C (HCV RNA positive), 5 (3.9%) had acute hepatitis E (IgM anti-HEV positive), and the other 53 (41.4%) were presumably acute hepatitis non-A-E. The prevalence of acute hepatitis A, B, E, and non-A-E showed no significant sex difference, whereas acute hepatitis C was significantly more prevalent in females. The prevalence of acute hepatitis A and B decreased and that of acute hepatitis C increased significantly with increasing age. In contrast, acute hepatitis E and non-A-E showed no significant age predominance. Of 177 HBsAg carriers with acute hepatitis, 64 (36.1%) demonstrated non-B hepatotropic virus superinfection, with HCV being the most common (60.9%), followed by hepatitis D, E, and A viruses, and the other 55 (31.1%) and 58 (32.8%) were presumed to have acute exacerbation of chronic hepatitis B or superimposed acute hepatitis non-A-E, respectively. Serum GBV-C/HGV RNA was detected in 3-4% of acute hepatitis non-A-E cases, suggesting its limited role in these cases.
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| pubmed:language |
eng
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| pubmed:journal | |
| pubmed:citationSubset |
IM
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| pubmed:chemical | |
| pubmed:status |
MEDLINE
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| pubmed:month |
Jun
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| pubmed:issn |
0146-6615
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| pubmed:author | |
| pubmed:issnType |
Print
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| pubmed:volume |
58
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| pubmed:owner |
NLM
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| pubmed:authorsComplete |
Y
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| pubmed:pagination |
154-9
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| pubmed:dateRevised |
2006-11-15
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| pubmed:meshHeading |
pubmed-meshheading:10335863-Acute Disease,
pubmed-meshheading:10335863-Adolescent,
pubmed-meshheading:10335863-Adult,
pubmed-meshheading:10335863-Age Distribution,
pubmed-meshheading:10335863-Aged,
pubmed-meshheading:10335863-Aged, 80 and over,
pubmed-meshheading:10335863-Carrier State,
pubmed-meshheading:10335863-Endemic Diseases,
pubmed-meshheading:10335863-Female,
pubmed-meshheading:10335863-Flaviviridae,
pubmed-meshheading:10335863-Hepacivirus,
pubmed-meshheading:10335863-Hepatitis, Viral, Human,
pubmed-meshheading:10335863-Hepatitis A,
pubmed-meshheading:10335863-Hepatitis B,
pubmed-meshheading:10335863-Hepatitis B Surface Antigens,
pubmed-meshheading:10335863-Hepatitis E virus,
pubmed-meshheading:10335863-Humans,
pubmed-meshheading:10335863-Male,
pubmed-meshheading:10335863-Middle Aged,
pubmed-meshheading:10335863-RNA, Viral,
pubmed-meshheading:10335863-Taiwan
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| pubmed:year |
1999
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| pubmed:articleTitle |
Etiology of sporadic acute viral hepatitis in Taiwan: the role of hepatitis C virus, hepatitis E virus and GB virus-C/hepatitis G virus in an endemic area of hepatitis A and B.
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| pubmed:affiliation |
Liver Research Unit, Chang Gung Memorial Hospital, Chang Gung University, Taipei, Taiwan. gi31208108@adm.cgmh.com.tw
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| pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|