Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
16
pubmed:dateCreated
1999-5-17
pubmed:abstractText
Impaired endothelium-dependent, nitric oxide (NO)-mediated vasodilation may contribute to increased vasomotor tone in patients with heart failure. Whether decreased endothelium-dependent, NO-mediated vasodilation in patients with heart failure is due to decreased synthesis or increased degradation of NO is unknown.
pubmed:grant
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
27
pubmed:volume
99
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2113-7
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed-meshheading:10217650-Adult, pubmed-meshheading:10217650-Aged, pubmed-meshheading:10217650-Arginine, pubmed-meshheading:10217650-Cardiomyopathies, pubmed-meshheading:10217650-Creatinine, pubmed-meshheading:10217650-Exercise Test, pubmed-meshheading:10217650-Female, pubmed-meshheading:10217650-Heart Failure, pubmed-meshheading:10217650-Humans, pubmed-meshheading:10217650-Leg, pubmed-meshheading:10217650-Male, pubmed-meshheading:10217650-Middle Aged, pubmed-meshheading:10217650-Nitrates, pubmed-meshheading:10217650-Nitric Oxide, pubmed-meshheading:10217650-Nitrogen Isotopes, pubmed-meshheading:10217650-Reference Values, pubmed-meshheading:10217650-Regional Blood Flow, pubmed-meshheading:10217650-Urea, pubmed-meshheading:10217650-Ventricular Dysfunction, Left, pubmed-meshheading:10217650-Ventricular Function, Left
pubmed:year
1999
pubmed:articleTitle
Decreased activity of the L-arginine-nitric oxide metabolic pathway in patients with congestive heart failure.
pubmed:affiliation
Columbia Presbyterian Medical Center, Division of Circulatory Physiology, Department of Medicine, Columbia University College of Physicians and Surgeons, New York, NY, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S.