rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
15
|
pubmed:dateCreated |
1999-4-30
|
pubmed:abstractText |
ACE (kininase II) inhibitors have been shown to exert their beneficial cardiovascular effects via the inhibition of both angiotensin II formation and bradykinin breakdown. Because recent evidence suggests that ACE inhibitors may also interfere with B2 kinin receptor signaling and thus enhance the vascular response to bradykinin, we examined whether the distribution of B2 kinin receptors within the plasma membrane of native endothelial cells is affected by an ACE inhibitor.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Angiotensin-Converting Enzyme...,
http://linkedlifedata.com/resource/pubmed/chemical/Bradykinin,
http://linkedlifedata.com/resource/pubmed/chemical/Calcium-Calmodulin-Dependent...,
http://linkedlifedata.com/resource/pubmed/chemical/Membrane Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Mitogen-Activated Protein Kinase 1,
http://linkedlifedata.com/resource/pubmed/chemical/Mitogen-Activated Protein Kinase 3,
http://linkedlifedata.com/resource/pubmed/chemical/Mitogen-Activated Protein Kinases,
http://linkedlifedata.com/resource/pubmed/chemical/Ramipril,
http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Bradykinin B2,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Bradykinin,
http://linkedlifedata.com/resource/pubmed/chemical/icatibant,
http://linkedlifedata.com/resource/pubmed/chemical/ramiprilat
|
pubmed:status |
MEDLINE
|
pubmed:month |
Apr
|
pubmed:issn |
1524-4539
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:day |
20
|
pubmed:volume |
99
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
2034-40
|
pubmed:dateRevised |
2009-11-19
|
pubmed:meshHeading |
pubmed-meshheading:10209009-Angiotensin-Converting Enzyme Inhibitors,
pubmed-meshheading:10209009-Animals,
pubmed-meshheading:10209009-Aorta,
pubmed-meshheading:10209009-Biological Transport,
pubmed-meshheading:10209009-Bradykinin,
pubmed-meshheading:10209009-Calcium Signaling,
pubmed-meshheading:10209009-Calcium-Calmodulin-Dependent Protein Kinases,
pubmed-meshheading:10209009-Endothelium, Vascular,
pubmed-meshheading:10209009-Membrane Proteins,
pubmed-meshheading:10209009-Mitogen-Activated Protein Kinase 1,
pubmed-meshheading:10209009-Mitogen-Activated Protein Kinase 3,
pubmed-meshheading:10209009-Mitogen-Activated Protein Kinases,
pubmed-meshheading:10209009-Ramipril,
pubmed-meshheading:10209009-Receptor, Bradykinin B2,
pubmed-meshheading:10209009-Receptors, Bradykinin,
pubmed-meshheading:10209009-Signal Transduction,
pubmed-meshheading:10209009-Swine
|
pubmed:year |
1999
|
pubmed:articleTitle |
Angiotensin-converting enzyme inhibitor ramiprilat interferes with the sequestration of the B2 kinin receptor within the plasma membrane of native endothelial cells.
|
pubmed:affiliation |
Institut für Kardiovaskuläre Physiologie, Klinikum der J.W. Goethe-Universität, Frankfurt am Main, Germany.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|