Source:http://linkedlifedata.com/resource/pubmed/id/10200949
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
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pubmed:dateCreated |
1999-6-28
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pubmed:abstractText |
Anorexia and body weight loss are characteristic of many diseases, including cancer and AIDS. Recent studies indicate that inflammatory cytokines, such as interleukin 1, the interleukin 6 subfamily and tumor necrosis factor, induce anorexia and cachexia by inhibiting the normal adaptive feeding response to energy deficits. Here, I discuss the evidence for and against a central role for neuropeptide Y and leptin in anorexia and cachexia.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
1357-4310
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
5
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
79-85
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:10200949-Animals,
pubmed-meshheading:10200949-Anorexia,
pubmed-meshheading:10200949-Appetite,
pubmed-meshheading:10200949-Cachexia,
pubmed-meshheading:10200949-Cytokines,
pubmed-meshheading:10200949-Humans,
pubmed-meshheading:10200949-Hypothalamus,
pubmed-meshheading:10200949-Neuropeptide Y,
pubmed-meshheading:10200949-Syndrome,
pubmed-meshheading:10200949-Wasting Syndrome
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pubmed:year |
1999
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pubmed:articleTitle |
Neuropeptide Y: a key molecule in anorexia and cachexia in wasting disorders?
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pubmed:affiliation |
Second Dept of Internal Medicine, Kobe University School of Medicine, Japan. inui@med.kobe-u.ac.jp
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pubmed:publicationType |
Journal Article,
Review,
Research Support, Non-U.S. Gov't
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