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rdf:type |
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lifeskim:mentions |
umls-concept:C0011849,
umls-concept:C0020443,
umls-concept:C0020538,
umls-concept:C0028128,
umls-concept:C0030685,
umls-concept:C0079284,
umls-concept:C0332307,
umls-concept:C0391871,
umls-concept:C0680255,
umls-concept:C1280500,
umls-concept:C1283071,
umls-concept:C1882714,
umls-concept:C1963578
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pubmed:issue |
9
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pubmed:dateCreated |
1999-4-13
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pubmed:abstractText |
Although endothelin-1 is a potent vasoconstrictor peptide, stimulation of endothelin type B receptor (ETBR) causes bidirectional changes in vascular tone, ie, vasodilation and vasoconstriction. Roles of ETBR in pathological conditions are largely unknown.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/BQ 3020,
http://linkedlifedata.com/resource/pubmed/chemical/Endothelin-1,
http://linkedlifedata.com/resource/pubmed/chemical/Endothelins,
http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide,
http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase,
http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type III,
http://linkedlifedata.com/resource/pubmed/chemical/Nos3 protein, rat,
http://linkedlifedata.com/resource/pubmed/chemical/Peptide Fragments,
http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Endothelin B,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Endothelin
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0009-7322
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pubmed:author |
pubmed-author:GotoAA,
pubmed-author:HayakawaHH,
pubmed-author:HirataYY,
pubmed-author:KakokiMM,
pubmed-author:KikuchiKK,
pubmed-author:KimuraKK,
pubmed-author:NaganoTT,
pubmed-author:NagataDD,
pubmed-author:OmataMM,
pubmed-author:RottN NNN,
pubmed-author:SuzukiEE
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pubmed:issnType |
Print
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pubmed:day |
9
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pubmed:volume |
99
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1242-8
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:10069794-Animals,
pubmed-meshheading:10069794-Blood Pressure,
pubmed-meshheading:10069794-Diabetes Mellitus, Experimental,
pubmed-meshheading:10069794-Endothelin-1,
pubmed-meshheading:10069794-Endothelins,
pubmed-meshheading:10069794-Hypercholesterolemia,
pubmed-meshheading:10069794-Hypertension,
pubmed-meshheading:10069794-Immunohistochemistry,
pubmed-meshheading:10069794-Kidney,
pubmed-meshheading:10069794-Male,
pubmed-meshheading:10069794-Nitric Oxide,
pubmed-meshheading:10069794-Nitric Oxide Synthase,
pubmed-meshheading:10069794-Nitric Oxide Synthase Type III,
pubmed-meshheading:10069794-Peptide Fragments,
pubmed-meshheading:10069794-Rats,
pubmed-meshheading:10069794-Rats, Inbred Dahl,
pubmed-meshheading:10069794-Rats, Inbred SHR,
pubmed-meshheading:10069794-Rats, Inbred WKY,
pubmed-meshheading:10069794-Rats, Sprague-Dawley,
pubmed-meshheading:10069794-Receptor, Endothelin B,
pubmed-meshheading:10069794-Receptors, Endothelin,
pubmed-meshheading:10069794-Vascular Resistance
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pubmed:year |
1999
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pubmed:articleTitle |
Effects of hypertension, diabetes mellitus, and hypercholesterolemia on endothelin type B receptor-mediated nitric oxide release from rat kidney.
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pubmed:affiliation |
Second Department of Internal Medicine, Faculty of Medicine, University of Tokyo, Tokyo, Japan.
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
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