Source:http://linkedlifedata.com/resource/pubmed/id/10066504
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4
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pubmed:dateCreated |
1999-5-5
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pubmed:abstractText |
Recent research has brought additional information on how virus products interfere with host cell antigen processing in vitro, new information on the interaction of virus with dendritic cells as a mechanism for alteration of immune responses - especially immunosuppression, and a preliminary proposal that nonretroviral RNA viruses might persist by utilizing host-cell reverse transcriptase to enter a DNA phase of replication.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
1369-5274
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
1
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
436-41
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:10066504-Animals,
pubmed-meshheading:10066504-Antigen Presentation,
pubmed-meshheading:10066504-Dendritic Cells,
pubmed-meshheading:10066504-Humans,
pubmed-meshheading:10066504-Immune Tolerance,
pubmed-meshheading:10066504-RNA Viruses,
pubmed-meshheading:10066504-Virus Diseases,
pubmed-meshheading:10066504-Virus Replication
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pubmed:year |
1998
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pubmed:articleTitle |
Viral persistence: mechanisms and consequences.
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pubmed:affiliation |
Division of Virology, Department of Neuropharmacology, The Scripps Research Institute, 10550 North Torrey Pines Road, La Jolla, CA 92037, USA. mbaobo@scripps.edu
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Review,
Research Support, Non-U.S. Gov't
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