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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
1999-3-2
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pubmed:abstractText |
Preclinical and clinical studies suggest that lowered brain serotonin neurotransmission may contribute to the pathophysiology of bulimia nervosa (BN). The aim of our study was to test this hypothesis by examining the psychological effects of a dietary-induced impairment in serotonin activity in subjects known to be at risk for manifestation of the clinical syndrome of BN.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0003-990X
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
56
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
171-6
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pubmed:dateRevised |
2009-9-29
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pubmed:meshHeading |
pubmed-meshheading:10025442-Acute Disease,
pubmed-meshheading:10025442-Adult,
pubmed-meshheading:10025442-Affect,
pubmed-meshheading:10025442-Amino Acids,
pubmed-meshheading:10025442-Body Image,
pubmed-meshheading:10025442-Bulimia,
pubmed-meshheading:10025442-Cross-Over Studies,
pubmed-meshheading:10025442-Depressive Disorder,
pubmed-meshheading:10025442-Diet,
pubmed-meshheading:10025442-Double-Blind Method,
pubmed-meshheading:10025442-Feeding Behavior,
pubmed-meshheading:10025442-Female,
pubmed-meshheading:10025442-Humans,
pubmed-meshheading:10025442-Psychiatric Status Rating Scales,
pubmed-meshheading:10025442-Recurrence,
pubmed-meshheading:10025442-Serotonin,
pubmed-meshheading:10025442-Tryptophan
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pubmed:year |
1999
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pubmed:articleTitle |
Symptomatic relapse in bulimia nervosa following acute tryptophan depletion.
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pubmed:affiliation |
University Department of Psychiatry, Warneford Hospital, Oxford, England.
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pubmed:publicationType |
Journal Article,
Clinical Trial,
Comparative Study,
Controlled Clinical Trial,
Research Support, Non-U.S. Gov't
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