| Predicate | Object |
|---|---|
| rdf:type | |
| entrezgene:geneRifText |
Heteromeric Kir2.1-V227F and WT channels showed an unusual latent loss of function biophysical phenotype that depended on PKA-dependent Kir2.1 phosphorylation, distinct from typical Andersen-Tawil mutations.
|