| http://linkedlifedata.com/r... | rdf:type | entrezgene:Interaction | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19100722 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:22114277 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:9540970 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:12972513 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:interactant | entrez-gene:155971 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:geneRifText | HIV-1 gp120-induced neuron death is inhibited by CCL3L1 treatment via the phosphorylation of CREB, the upregulation of Bcl-2, and the PKA and CaMKI/CaMKIV cell signaling pathways | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:geneRifText | HIV-1 gp120 activates forward trafficking and surface clustering of NMDA receptors in membrane microdomains by a PKA-dependent phosphorylation of the NR1 C-terminal Ser897, followed by a PKC-dependent phosphorylation of Ser896 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:geneRifText | Treatment of normal human fibroblasts with HIV-1 gp120 protein results in a significant decrease in the levels of 8-azido-[32P]cAMP binding to RI and RII domains of protein kinase A (PKA) and in PKA activity | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:geneRifText | The exposure of CD4+ T cells to HIV gp120 results in a long-lasting hyporesponsiveness to antigen stimulation through protein kinase A-mediated signaling | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:keyphrase | interacts with | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:keyphrase | inhibited by | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:keyphrase | inactivates | lld:entrezgene |
| entrez-gene:5566 | entrezgene:interaction | http://linkedlifedata.com/r... | lld:entrezgene |
