| pubmed-article:9774280 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9774280 | lifeskim:mentions | umls-concept:C0178453 | lld:lifeskim |
| pubmed-article:9774280 | lifeskim:mentions | umls-concept:C0087071 | lld:lifeskim |
| pubmed-article:9774280 | lifeskim:mentions | umls-concept:C0038952 | lld:lifeskim |
| pubmed-article:9774280 | lifeskim:mentions | umls-concept:C1513371 | lld:lifeskim |
| pubmed-article:9774280 | pubmed:issue | 5388 | lld:pubmed |
| pubmed-article:9774280 | pubmed:dateCreated | 1998-10-30 | lld:pubmed |
| pubmed-article:9774280 | pubmed:abstractText | Deletion of the telomerase catalytic subunit gene trt1+ in Schizosaccharomyces pombe results in death for the majority of cells, but a subpopulation survives. Here it is shown that most survivors have circularized all of their chromosomes, whereas a smaller number maintain their telomeres presumably through recombination. When the telomeric DNA-binding gene taz1+ is also deleted, trt1- taz1- survivors use the recombinational mode more frequently. Moreover, the massive elongation of telomeres in taz1- cells is absent in the double mutant. Thus, Taz1p appears to regulate telomeric recombination as well as telomerase activity in fission yeast. | lld:pubmed |
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| pubmed-article:9774280 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9774280 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9774280 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9774280 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:9774280 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:9774280 | pubmed:author | pubmed-author:CechT RTR | lld:pubmed |
| pubmed-article:9774280 | pubmed:author | pubmed-author:CooperJ PJP | lld:pubmed |
| pubmed-article:9774280 | pubmed:author | pubmed-author:NakamuraT MTM | lld:pubmed |
| pubmed-article:9774280 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9774280 | pubmed:day | 16 | lld:pubmed |
| pubmed-article:9774280 | pubmed:volume | 282 | lld:pubmed |
| pubmed-article:9774280 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9774280 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9774280 | pubmed:pagination | 493-6 | lld:pubmed |
| pubmed-article:9774280 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:9774280 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9774280 | pubmed:articleTitle | Two modes of survival of fission yeast without telomerase. | lld:pubmed |
| pubmed-article:9774280 | pubmed:affiliation | Howard Hughes Medical Institute, Department of Chemistry and Biochemistry, University of Colorado, Boulder, CO 80309-0215, USA. | lld:pubmed |
| pubmed-article:9774280 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9774280 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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